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Study to Evaluate the Association of Testosterone Levels With Coronary Artery Calcification

Conditions
Coronary Artery Disease
Interventions
Other: No intervention
Registration Number
NCT02310659
Lead Sponsor
Zhejiang University
Brief Summary

Coronary artery calcification (CAC) is a pandemic condition in elderly patients with coronary artery disease (CAD) and associated with worse prognosis. Although available data shows association between testosterone levels in men and CAD, the association between testosterone and CAC in old-aged male patients with CAD remains unknown. In this study, the relationship of serum testosterone levels with CAC score in elderly male patients with CAD was evaluated.

Detailed Description

Coronary artery calcification (CAC) is associated with worse outcomes in patients with coronary artery disease (CAD), especially in old-aged population. Extensive CAC also increases the risk of procedure associated complications, such as stent migration, coronary artery perforation, dissection and thrombosis. The pathogenesis of CAC has not been fully explained. Recent evidence suggests that CAC share common pathways with bone formation. So, it can be presumed that risk factors contributing to bone formation and resorption activity also affect the development of CAC. Sex hormones is known to play an important role in bone development and in bone quality maintenance. Available data shows that androgens may affect differentiation, proliferation, and apoptosis of osteocytes, osteoclasts, and osteoblasts. Androgen receptor expression has been detected on different types of cells which contributing to the bone formation, such as osteoblastes, osteocytes and condrocytes, and androgen deprivation therapy results negative effects on bone mineral density in patients with metastatic prostate cancer. Osteoporosis, a systemic disease characterized by bone tissue loss, is more prevalent in oler men with low testosterone levels. In CAC, pathophysiological researches show that several vascular cell types, such as vascular smooth muscle cells, adventitial myofibroblasts and microvascular pericytes, have the potential to produce mineralized matrix and differentiate into osteoblasts. So, it can be postulated that androgens may play a similar role in the development of CAC.

Up to date, only limited data is available about the association between testosterone and CAC. A single-center study with relatively small sample revealed an inverse association between testosterone and CAC in non-obese men. The aim of this study was to investigate the association between testosterone and CAC measured as CAC score in old-aged male patients with CAD.

Recruitment & Eligibility

Status
UNKNOWN
Sex
Male
Target Recruitment
200
Inclusion Criteria
  • Elderly male patients (age ≥ 65 years old) with stable angina and proven coronary artery disease
Exclusion Criteria
  • Less than 65 years old
  • Acute coronary syndrome
  • History of PCI or coronary artery bypass grafting
  • History of myocardial infarction
  • Renal dysfunction defined by glomerular filtration rate < 30 mL/min

Study & Design

Study Type
OBSERVATIONAL
Study Design
Not specified
Arm && Interventions
GroupInterventionDescription
lower calcificant score groupNo interventionpatients with coronary artery calcificant score \<400
higher calcificant score groupNo interventionpatients with coronary artery calcificant score ≥400
Primary Outcome Measures
NameTimeMethod
Number of participants with severe coronary artery calcification10 months

Number of participants with coronary artery calcification score ≥400

Secondary Outcome Measures
NameTimeMethod

Trial Locations

Locations (1)

1st Affiliated Hospital, College of Medicine, Zhejiang University

🇨🇳

Hangzhou, Zhejiang, China

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