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Short-term Fasting as an Enhancer of Chemotherapy: Pilot Clinical Study on Colorectal Carcinoma Patients

Not Applicable
Completed
Conditions
Fasting
Interventions
Procedure: Fasting
Registration Number
NCT04247464
Lead Sponsor
IMDEA Food
Brief Summary

This study will evaluate the ability of short-term fasting to reduce chemotherapy toxicity and enhance anti-tumour response in patients with colorectal carcinoma subjected to chemotherapy.

Detailed Description

Fasting for 24-48 hours during chemotherapy improves the response of the immune system against tumors and reduces chemotherapy toxicity through yet unknown mechanisms. The investigators have found that fasting induces the activation of p21, a protein that stops cell proliferation and plays important immune roles. The investigators hypothesize that p21 induction with short-term fasting enhances the immune anti-tumour response and reduces chemotherapy toxicity. To test this, half of the colorectal carcinoma (CRC) participants will follow 48 hours of fasting, 24 before and 24 after chemotherapy, under constant and specialized nutritional supervision. While the other half will follow a standard diet. A complete blood immunological profile at each chemotherapy cycle will be generated in collaboration with expert cytometrists, and gene expression, biochemical parameters, tumor evolution and toxicity markers will be measured. The investigators will (1) perform a complete analysis of immune cells to characterize the immune effects of fasting during chemotherapy; (2) analyze the effects of fasting on genes, metabolites and other molecules, to identify the responsible biological mechanisms, focusing on p21; (3) assess the reduction of chemotherapy toxicity in patients of colorectal carcinoma subjected to short-term fasting during chemotherapy.

Our project will further explore a safe, inexpensive, relatively unexplored and powerful nutritional intervention that can improve the quality of life and survival rates of millions of cancer patients: short-term fasting. Also, our project will have an important scientific impact, since previous reports have not yet described a clear mechanism explaining the beneficial effects of short-term fasting with chemotherapy

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
11
Inclusion Criteria
  • Participants with malignant colorectal neoplasia
  • Good metabolic state (BMI>22)
  • Good nutritional tests
  • Normal Haematological and biochemical parameters
  • Normal renal and hepatic function
  • No loss of weight during the chemotherapy treatment
Exclusion Criteria
  • BMI<22
  • Pregnancy or lactating women
  • Bad nutritional state
  • 3% weigh loss during the last month or more than 5% in the last three months
  • Diagnosis of type 2 diabetes mellitus or hypertension
  • Diagnosed hepatic, renal or cardiovascular disease
  • Respiratory of psychiatric disease
  • Nausea or vomiting, gastrointestinal disease

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
FastingFastingThe participants will follow a short-term fasting period for 44-48 hours, starting 24 hours before chemotherapy treatment
Primary Outcome Measures
NameTimeMethod
Changes in the immune responseBaseline and after three weeks

To evaluate the effect of short-term fasting on the immune response a complete immune phenotyping by flow cytometry will be done: cluster of differentiation 3 (CD3), cluster of differentiation 4 (CD4), cluster of differentiation 8 (CD8) (for T cells); cluster of differentiation 19 (CD19) (for B-cells), the high affinity Interleukin-2 receptor alpha subunit (CD45RA), CD62L (for T cell subsets: Memory, Effector); cluster of differentiation 25 (CD25) and cluster of differentiation 127 (CD127) (both for Treg cells); cluster of differentiation 11b C(D11b) (for granulocytes and macrophages); cluster of differentiation 14 (CD14) (for monocytes); cluster of differentiation antigen 16 (CD16), cluster of differentiation 56 (CD56) (NK cells); cluster of differentiation 15 (CD15) (for granulocytes and monocytes) markers will be analyzed

Changes in the correlation between chemotherapy response and p21 and/or other fasting genes expression in peripheral blood mononuclear cells (PBMCs)Baseline and after three weeks

The expression levels of p21 and/or fasting genes in peripheral blood mononuclear cells (PBMCs) will be correlated with toxicity parameters previously described in the primary outcome measure 1

Changes in the Common Terminology Criteria for Adverse Events CTCAE 5.0 toxicity table score.Baseline and after three weeks

To evaluate changes in chemotherapy toxicity, the Common Terminology Criteria for Adverse Events (CTCAE) 5.0 toxicity table score will be calculated, taking into account different analysis and questionnaires on toxicity symptoms.

Analysis will include:

* Hematological analysis (erythrocytes, thrombocytes, white blood cells, Neutrophil/lymphocyte ratio and Platelet/lymphocyte ratio).

* Biochemical analysis (sodium, potassium, calcium, phosphate, urea, creatinine, total protein, albumin, bilirubin, alkaline phosphatase, lactate dehydrogenase, alanine transaminase, aspartate transaminases, creatine kinase, troponin T, C Reactive Protein (CRP), cortisol and prealbumin)

* Subjective symptoms obtained from health questionnaires (hunger, nausea, dizzying, weakness, diarrhea, constipation, gastroesophageal reflux disease)

Secondary Outcome Measures
NameTimeMethod
Subjective evaluation of tolerance to fasting48 hours of fasting, including 24 hours prior and 24 hours after chemotherapy administration.

To evaluate the tolerance to fasting, participants will fill in a fasting tolerance test based on the symptoms they feel, this will result in a final score of tolerance to fasting.

Changes in Insulin levels in response to fastingBaseline and after three weeks

Insulin levels (International Units per milliliter) will be measured with a kit from Abbott Laboratories, by luminescent immunoassay using the Architect instrument from Abbott Laboratories.

Changes ketone bodies in response to fastingBaseline and after three weeks

Ketone bodies concentration (moles per milliliter) will be measured with a kit from Sigma-Aldrich, by an enzymatic spectrophotometric assay using an microplate reader from Thermo Fisher.

Antitumoral response associated to fasting after chemotherapy treatmentBaseline and after three weeks

To evaluate the clinical antitumoral response, different tumoral markers such as carcinoembryonic antigen (CEA) and Carbohydrate antigen (Ca 19.9) will be analyzed in serum samples

Changes in glycemia in response to fastingBaseline and after three weeks

Glucose levels (milligrams per milliliter) will be measured with a kit from Abbott Laboratories, by enzymatic spectrophotometric assays using an Architect instrument from Abbot Laboratories.

Changes in Free Fatty Acids levels in response to fastingBaseline and after three weeks

Free fatty acids levels (moles per milliliter) will be evaluated with a kit from Abbott Laboratories, by enzymatic spectrophotometric assays using an Architect instrument from Abbott Laboratories.

Changes in gene expression in PBMCs after fastingBaseline and after three weeks

To evaluate changes in gene expression in PBMCs the following fasting genes will be analyzed by qRTPCR:

* p21

* Pyruvate Dehydrogenase Kinase 4 (PDK4)

* Carnitine palmitoyltransferase 1 (CPT1)

* Adipophilin (ADFP)

* Solute carrier family 25, member 50 (SLC25A50)

Trial Locations

Locations (1)

IMDEA Food

🇪🇸

Madrid, Spain

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