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First-in-Human Study of TSHA-101 Gene Therapy for Treatment of Infantile Onset GM2 Gangliosidosis

Phase 1
Active, not recruiting
Conditions
Infantile GM2 Gangliosidosis (Disorder)
Interventions
Biological: TSHA-101
Registration Number
NCT04798235
Lead Sponsor
Dr. Anupam Sehgal
Brief Summary

GM2 gangliosidoses are a group of autosomal recessive neurodegenerative diseases characterized by a deficiency of the Hex A enzyme to catabolize GM2, thereby causing GM2 accumulation within cellular lysosomes.Hex A is composed of 2 subunits, α- and β-, coded by the HEXA and HEXB genes, respectively. The primary purpose of the current study is to assess the safety and tolerability of TSHA101 administered via IT injection.

Detailed Description

Not available

Recruitment & Eligibility

Status
ACTIVE_NOT_RECRUITING
Sex
All
Target Recruitment
3
Inclusion Criteria
  • male or female with age less than or equal to 15 months
  • diagnosis of GM2 gangliosidosis with genetic and enzymatic documentation of infantile disease

Key

Exclusion Criteria
  • a second neurodevelopmental disorder independent of the HEXA or HEXB
  • inability to tolerate sedation or intrathecal administration
  • invasive ventilatory support
  • concomitant illness, allergies or known hypersensitivity to the required immunosuppression regimen

Study & Design

Study Type
INTERVENTIONAL
Study Design
SINGLE_GROUP
Arm && Interventions
GroupInterventionDescription
TSHA-101TSHA-101Subjects who will receive one-time intrathecal TSHA-101, brain volume based sliding scale for dosage
Primary Outcome Measures
NameTimeMethod
Safety and Tolerability: Number of participants with abnormal Laboratory assessments1 year

Number of participants with Changes from Baseline in laboratory assessments

Safety and tolerability: Treatment-emergent Adverse Events (TEAEs)1 year

Incidence, severity, and relatedness of TEAEs

Safety and Tolerability: Electrocardiogram (ECG)1 year

Changes from Baseline in 12-lead ECG findings in QT interval

Secondary Outcome Measures
NameTimeMethod
Assessment of Immunogenicity: Biomarkers in serum1 year

Summary of total antibodies (TAbs) titers for AAV9 and Hex A

Assessment of Immunogenicity: Biomarkers in peripheral blood mononuclear cells (PBMCs5 years

Summary of PBMCs for enzyme-linked immune absorbent spot (ELISpot) assays for cytokine secretion against AAV9 and Hex A

Safety and tolerability: Viral shedding analysis1 year

Positive presence of viral DNA from biological fluids (whole blood, urine, saliva, and stool)

Overall Survivaltreatment to death from any cause, up to 5 years

Estimated using the Kaplan-Meier method

Hex A Enzyme Activity: Cerebrospinal fluid (CSF) and serum1 year

Change from baseline

Head Control: Number of events for abnormal head control1 year

change from Baseline

Change from Baseline in motor function: Children's Hospital of Philadelphia Infant Test of Neuromuscular Disorders (CHOP-INTEND)1 year

The test consists of 16 items (body parts), where each item is tested for both sides of the body, left and right. The best score is taken for each item (with a maximum score of 4), and the scores are summed over all 16 items with a possible total CHOP-INTEND score of 64.

Change from Baseline in Motor Function: Modified Ashworth Scale1 year

change from Baseline. Increase or decrease of muscle tone will be measured by the Modified Ashworth Scale. Frequency counts and percentages will be presented by score (0, 1, 1+, 2, 3, and 4), muscle, side, and visit for the safety population. Flexion and extension of the knee and elbow will be measured on both sides, along with hip adduction and abduction on both sides of the body.

Clinical Efficacy Assessment: Progression of Hypotonia1 year

Assessed through neurological examinations as present or absent. Baseline to each post-Baseline visit

Clinical Efficacy Assessment: DysphagiaFrom onset up to 3 years, if present

Assessment of the dysphagia events- assessed as present or absent.

Trial Locations

Locations (1)

Queen's University/Kingston Health Sciences Centre

🇨🇦

Kingston, Ontario, Canada

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