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Liver Health and Metabolic Function in People With Obesity

Not Applicable
Conditions
Non-Alcoholic Fatty Liver Disease
Interventions
Procedure: Weight loss surgery
Registration Number
NCT03701828
Lead Sponsor
Washington University School of Medicine
Brief Summary

This project aims to determine the effect of significant weight loss on rates on hepatic fibrogenesis in people with obesity.

Detailed Description

Non-alcoholic fatty liver disease (NAFLD) affects approximately 70% of those with obesity and if left untreated can progress to cirrhosis and liver failure. NAFLD stages progress from the stage of simple steatosis to steatohepatitis, and fibrosis The stage of NAFLD is currently best determined using histology from a liver biopsy, however this provides a static depiction of the state of the liver. Therefore, this project aims to determine fibrogenesis in the liver of obese humans in vivo, before and after weight loss.

Recruitment & Eligibility

Status
ENROLLING_BY_INVITATION
Sex
All
Target Recruitment
30
Inclusion Criteria
  • Body mass index ≥35 kg/m2
  • Scheduled for Bariatric surgery
Read More
Exclusion Criteria
  • Significant organ disfunction/disease
  • Tobacco use
  • Previous bariatric surgery
  • Pregnancy
  • Excessive alcohol use
  • Liver disease other than NAFLD
Read More

Study & Design

Study Type
INTERVENTIONAL
Study Design
SINGLE_GROUP
Arm && Interventions
GroupInterventionDescription
Weight lossWeight loss surgeryParticipants will undergo weight loss surgery
Primary Outcome Measures
NameTimeMethod
Liver histologyBefore and after ~20% weightloss, up to 6 months

Analysis of liver tissue morphology

Hepatic FibrogenesisBefore and after ~20% weightloss, up to 6 months

Rates of liver collagen synthesis

Secondary Outcome Measures
NameTimeMethod
Adipose histologyBefore and after ~20% weightloss, up to 6 months

Assessment of adipose tissue morphology

Tissue gene expressionBefore and after ~20% weightloss, up to 6 months

Messenger ribonucleic acid content of adipose, liver and blood tissues will be assessed

Plasma protein synthesisBefore and after ~20% weightloss, up to 6 months

Synthesis rates of collagen-related proteins in plasma

Tissue inflammationBefore and after ~20% weightloss, up to 6 months

adipose tissue and blood immune cell content

Tissue bacterial contaminationBefore and after ~20% weightloss, up to 6 months

bacterial 16s ribosomal ribonucleic acid content will be assessed in blood and tissue samples

Body compositionBefore and after ~20% weightloss, up to 6 months

Body composition assessed by dual-energy x-ray absorptiometry and magnetic resonance scans

Intestinal permeabilityBefore and after ~20% weightloss, up to 6 months

Urinary excretion of indigestible sugars will be assessed over 24 hours after an oral challenge

Tissue lipidomicsBefore and after ~20% weightloss, up to 6 months

bioactive lipid content of adipose, liver and blood tissues will be assessed

tissue extracellular vesiclesBefore and after ~20% weightloss, up to 6 months

amount, content and function of extracellular vesicles obtained from adipose, liver, and blood tissue

Liver extracellular vesiclesat the time of surgery only

amount, content and function of extracellular vesicles obtained from liver tissue

Systemic inflammationBefore and after ~20% weightloss, up to 6 months

Plasma cytokine concentrations

Insulin sensitivityBefore and after ~20% weightloss, up to 6 months

Whole-body insulin sensitivity during a hyperinsulinemic-euglycemic clamp

adipose tissue progenitor cellsBefore and after ~20% weightloss, up to 6 months

content and function of progenitor cells obtained from adipose tissue

Liver single cell RNA seqat the time of surgery only

gene expression of liver immune cell types

Liver flow cytometryat the time of surgery only

liver immune cell content

Trial Locations

Locations (1)

Washington University School of Medicine

🇺🇸

Saint Louis, Missouri, United States

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