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Urban Environmental Factors and Childhood Asthma

Completed
Conditions
Asthma
Allergy
Registration Number
NCT00114881
Lead Sponsor
National Institute of Allergy and Infectious Diseases (NIAID)
Brief Summary

Minority children who grow up in poor urban neighborhoods have the highest rates of asthma, and also experience greater morbidity from acute exacerbations of this disease. The aim of this study is to further identify environmental factors unique to the inner city that affect immune development and the expression of wheezing, atopy and asthma for purposes of identifying new strategies for asthma prevention.

Detailed Description

The purpose of this study is to determine the way environmental factors (like the components of inner-city household dust) affect immune system development and symptoms of asthma in inner city children. The study is divided into five periods, as the subjects age from birth to 17 years old. Each age bracket will explore different objectives and endpoints.

Study Objectives/Hypotheses:

1. Subjects age 0 to 3 years old:

* Environmental factors in the inner city adversely influence the development of the immune system to promote cytokine dysregulation, allergy, and recurrent wheezing by age 3.

* Children who have had a viral lower respiratory infection and have developed cytokine dysregulation by age 3 are at increased risk for the development of asthma by age 6.

2. Subjects age 4 to 7 years old:

* There is a unique pattern of immune development that is driven by specific urban exposures in early life, and this pattern of immune development is characterized by: 1) impairment of antiviral responses and 2) accentuation of Th2-like responses (e.g. cockroach-specific Interleukin-13(IL-13)). The clinical effects of these changes in immune development are frequent virus-induced wheezing and allergic sensitization by 3-4 years of age, and these characteristics synergistically increase the risk of asthma at age 7 years.

3. Subjects age 7 to 10 years old:

* There are unique combinations of environmental exposures (cockroach allergens, indoor pollutants \[Environmental Tobacco Smoke (ETS) and Nitrogen Dioxide (NO2)\], lack of microbial exposure), and family characteristics (stress, genetic factors related to innate immunity) that synergistically promote asthma onset, persistence, and morbidity in urban neighborhoods. These exposures and characteristics influence immune expression and lung development during critical periods of growth, resulting in specific asthma phenotypes.

4. Subjects age 10 to 16 years old:

-To determine the wheezing, asthma and atopy phenotypes in minority children growing up in poor urban neighborhoods as they develop from birth through adolescence.

5. Subjects to age 17 (Continuation of phase 4 to follow participants to age 17) To determine the wheezing, asthma and atopy phenotypes in minority children growing up in poor urban neighborhoods as they develop from birth through adolescence.

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
560
Inclusion Criteria

Not provided

Exclusion Criteria

Not provided

Study & Design

Study Type
OBSERVATIONAL
Study Design
Not specified
Primary Outcome Measures
NameTimeMethod
Development of wheezing0 to 3 years of age

Establish in inner city children the immunologic causes for the development of recurrent wheezing.

Correlation of Immunologic Factors and Development of Asthmaby 7 years of age

Establish, in this cohort of inner-city children, the immunologic causes for the development of asthma at age 7

Occurrence of Specific Phenotypes of Asthmaup to 17 years of age

Further define asthma phenotypes based on the findings in Inner-city Asthma Consortium-19 (ICAC-19) (Asthma Phenotypes in the Inner City (APIC), ClinicalTrials.gov Identifier NCT01383941).

Correlation of Risk Factors to Rapidly Evolving Asthma Phenotypesup to 10 years of age

Fully define the rapidly evolving asthma phenotypes and further delineate the role of risk factors related to environmental exposure (e.g.; house dust levels found through home inspection), immune development, lung growth on the natural history of asthma and allergic diseases in urban minority children

Incidence of Asthmaup to 17 years of age

Number of participants with the incidence (development) of asthma

Secondary Outcome Measures
NameTimeMethod

Trial Locations

Locations (4)

Boston Medical Center

🇺🇸

Boston, Massachusetts, United States

Pediatric Clinical Research Unit, Johns Hopkins University

🇺🇸

Baltimore, Maryland, United States

Columbia University Medical Center

🇺🇸

New York, New York, United States

Saint Louis Children's Hospital

🇺🇸

Saint Louis, Missouri, United States

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