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Evaluation of Biomarkers of COPD Exacerbation

Conditions
SAA Level and SAA/Lipoxin A4 Ratio
Registration Number
NCT03609541
Lead Sponsor
Heidelberg University
Brief Summary

Serum amlyoid A (SAA) was shown to act as biomarker for exacerbation of chronic obstructive pulmonary disease (AE-COPD). It seems that SAA triggers chronic inflammation by binding to ALX/PFR2 receptor. In contrast, lipoxin A4 seems to inhibit the inflammatory processes by binding to ALX/PFR2 receptor. A small trial has already demonstrated an imbalance between SAA and lipoxin A4 during AE-COPD. This study evaluates SAA level and SAA/lipoxin A4 ratio in patients with stable COPD and AE-COPD.

Detailed Description

Patient enrolment and data aquisition is to be carried out on a prospective basis. It is planned to enrol 40 patients with stable COPD and 40 patients with AE-COPD. All patients will undergo blood sampling inlcuding SAA and Lipoxin A4.

Recruitment & Eligibility

Status
UNKNOWN
Sex
All
Target Recruitment
80
Inclusion Criteria
  • Patients with COPD GOLD 3-4 confirmed by anamnesis and pulmonary function test
  • Patient has provided written informed consent
Exclusion Criteria
  • other reasons for worsening of symptoms (cough, dyspnea): e.g. pneumothorax, pneumonia, pulmonary embolism, myocardial infarction.
  • malignant disease

Study & Design

Study Type
OBSERVATIONAL
Study Design
Not specified
Primary Outcome Measures
NameTimeMethod
SAA/lipoxin A4 ratiotime of exacerbation/hospitalisation, an average of 3 days

SAA/lipoxin A4 ratio in stable COPD and COPD exacerbation

SAAtime of exacerbation/hospitalisation, an average of 3 days

SAA level in stable COPD and COPD exacerbation

Secondary Outcome Measures
NameTimeMethod

Trial Locations

Locations (1)

Thoraxklinik Heidelberg

🇩🇪

Heidelberg, Germany

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