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Corticosteroid Treatment in the Acute Phase of Caustic Ingestion Management

Phase 2
Conditions
Caustic Esophageal Injury
Esophageal Stenosis
Pharyngeal Stenosis
Interventions
Registration Number
NCT03760354
Lead Sponsor
Assistance Publique - Hôpitaux de Paris
Brief Summary

The management of patients who have ingested a caustic product has changed since 2007. Whereas previously the lesion assessment and surgical indication were based on endoscopic data, the therapeutic algorithm is currently based solely on the results of a CT scan with contrast injection, performed 6 hours after ingestion. This examination makes it possible to reliably assess the viability of the esophageal and gastric walls and thus to indicate digestive resection. The therapeutic consequences of this new treatment are important because, by expanding the indications for conservative treatment after severe ingestion, it brings a significant gain in terms of survival, morbidity and functional outcome. In the absence of emergency digestive resection, however, the functional prognosis is often overshadowed by the formation of esophageal stenosis in the months following ingestion. Patients then require endoscopic dilation treatment. In the event of failure or impossibility of dilation, which defines refractory stenosis, esophageal reconstruction is necessary. In case of sequential pharyngeal stenosis following ingestion, esophageal and pharyngeal reconstruction is indicated as a first-line treatment, since these stenosis do not respond to endoscopic dilations. The expansion of the indications for conservative treatment after severe ingestion using CT scans has led to an increase in the incidence of after-effect stenosis.

We aim to develop a therapeutic approach that will prevent the development of refractory and pharyngeal esophageal stenosis. Indeed, there is currently no strategy that has proven effective in this regard in adults. The value of corticosteroid therapy for the prevention of caustic stenosis has only been evaluated in children and remains controversial.

The main objective is to evaluate the effect of early systemic corticosteroid therapy on the risk of refractory esophageal or pharyngeal stenosis within one year of ingestion of a caustic substance in a population of patients at high risk of stenosis, defined according to tomodensitometric criteria (grade IIb: severe lesions, absence of transparietal necrosis), and for whom there is no indication of urgent digestive resection.

Detailed Description

Not available

Recruitment & Eligibility

Status
UNKNOWN
Sex
All
Target Recruitment
30
Inclusion Criteria

Not provided

Exclusion Criteria
  • Indication of resection or surgical exploration in emergency
  • History of caustic ingestion
  • Corticosteroids taken at a dose greater than 20 mg prednisone within 7 days before randomization
  • Contraindication to corticosteroid therapy:
  • Any infectious condition that required antibiotic treatment within 7 days of randomization
  • Any vaccine living within 7 days of randomization
  • Hypersensitivity to one of the components
  • Pregnancy in progress
  • Breastfeeding in progress
  • Co-intoxication involving vital prognosis and requiring, according to the patient's doctor, intensive care management
  • Patient under guardianship or curatorship

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Placebo (no corticosteroid treatment)PlaceboNaCl 0.9%, 100ml per day as a single slow intravenous administration over 60 minutes for a total treatment duration of 5 days
MethylprednisoloneMethylprednisoloneDose: 500mg/day for the first two days then 2mg/kg per day for three days. Dilution in 0.9% NaCl, 100 ml as a single slow intravenous administration over 60 minutes Total processing time of 5 days
Primary Outcome Measures
NameTimeMethod
Indication for esophageal or pharyngeal surgical reconstructionwithin 12 months post ingestion

The primary outcome is the indication for esophageal or pharyngeal surgical reconstruction due to:

1. The occurrence of one or more esophageal stenosis refractory to endoscopic dilation within 12 months of ingestion, as defined by:

* The need for more than 5 sessions of esophageal endoscopic dilation

* Non-expandable stenosis or esophageal obstruction;

* An esophageal perforation that occurs during dilation, which contraindicates the continuation of dilation.

2. The occurrence of pharyngeal stenosis, defined by the need to perform pharyngoplasty.

Secondary Outcome Measures
NameTimeMethod
Delay in the occurrence of refractory stenosis or pharyngeal stenosisat 9 months

Time between inclusion and occurrence of refractory stenosis or pharyngeal stenosis

Delay in the occurrence of refractory esophagal stenosis or pharyngeal stenosisat 12 months

Time between inclusion and occurrence of refractory stenosis or pharyngeal stenosis

Distance of the stenosisat 12 months

Distance between the stenosis and the dental arches (cm)

Number of stenosisat 12 months

Number of stenosis will be evaluated by endoscopy

Length of stenosisat 12 months

Length of each stenosis will be evaluated by endoscopy

Estimated diameter of stenosisat 12 months

Diameter of each stenosis will be evaluated by endoscopy

Endoluminal inflammationat 12 months

Importance of endoluminal inflammation during endoscopy performed for dilation (minimal, moderate, or severe)

Number of dilation sessionsat 12 months

Number of dilation sessions evaluated by endoscopy

Intervals between iterative dilationsat 12 months

Time between endoscopic dilatations if necessary iterative dilation

Extent of pharyngeal stenosisat 12 months

Laryngeal stenosis associated with pharyngeal stenosis

Digestive perforationsat 12 months

Proportion of digestive perforations secondary to endoscopic dilation

Proportion of unanticipated adverse reactionsat 12 months

Unanticipated adverse reactions will be defined by the occurrence of death,cardiac arrest whatever the cause,unplanned admission to intensive care unit (ICU) or extended stay \> 24 ICU

Proportion of adverse reactions related to corticosteroid therapywithin 7 days

Adverse reactions related to corticosteroid therapy will be defined by the occurrence of infections needing antibiotic therapy or spontaneous digestive perforation or digestive bleeding or cardiac arrhythmias de novo or pulmonary edema requiring treatment (increased oxygen requirements and/or Diuretic and/or Vasodilator and/or non-invasive ventilation and/or invasive ventilation) or respiratory complications (High blood pressure requiring treatment - Metabolic alkalosis- Hypokalemia\<3.0mmol/l - Delirium- Decompensation of a psychiatric pathology)

C-Reactive Protein (CRP)at one month

Inflammation markers

Tissue Growth Factor Beta (TGF beta)at one month

Fibrosis markers

interleukin-6 (IL-6)at one month

Inflammation markers

Tumour Necrosis Factor alpha (TNF alpha)at one month

Inflammation markers

interleukin-1 (IL1)at one month

Inflammation markers

Galectin 3at one month

Fibrosis markers

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