MedPath

Effects of Changing HIV Therapy at Lower Versus Higher Viral Loads

Phase 2
Completed
Conditions
HIV Infections
Registration Number
NCT00036465
Lead Sponsor
National Institute of Allergy and Infectious Diseases (NIAID)
Brief Summary

This study will look at people who have been taking anti-HIV drugs but still have detectable levels of HIV. The purpose of the study is to find out what happens in those people who change anti-HIV drugs when their viral load reaches 200 copies compared to those who change anti-HIV drugs when their viral load reaches 10,000 copies. This study will also look at drug resistance (how well HIV responds to drugs), viral fitness (how well drug-resistant HIV copies itself), and immunologic reconstitution (how well the immune system recognizes various infections, including HIV).

Many patients experience virologic relapse (increase in viral load after sustained viral load suppression) within 1 to 2 years of taking anti-HIV drugs. The approach to treatment for patients who experience virologic relapse while on a highly active antiretroviral therapy (HAART) has not been defined. Current guidelines recommend switching to a new treatment regimen as soon as possible to prevent HIV from becoming even more resistant to anti-HIV drugs. However, there is evidence that patients can benefit from staying on the same HAART drugs, even after virologic relapse. This study wants to find what happens when drugs are changed immediately after virologic relapse (when the viral load is lower) compared to what happens if drugs are changed only after a delay (when the viral load is higher).

Detailed Description

Virologic relapse occurs within 1 to 2 years of antiretroviral therapy in up to 50 percent of HIV-infected individuals. The best treatment approach for patients who experience virologic rebound while on highly active antiretroviral therapy (HAART) has not been defined. Current guidelines recommend switching to a new treatment regimen shortly after virologic rebound in an effort to avoid sequential accumulation of multiple resistance mutations. However, early treatment switching has numerous disadvantages: risk of virologic rebound on the new therapy, a limited number of drug combinations available to treat such rebounds, and difficulty in obtaining early genotypic and phenotypic drug-resistance information to guide treatment modification. Delaying a switch to a new antiretroviral regimen has the advantage of preserving future treatment options, and HIV levels may remain partially suppressed even after drug-resistant mutants emerge. Moreover, several observational studies describe maintenance of immunologic and clinical benefits of HAART even after virologic rebound. Delayed treatment switches, however, raise concerns about sequential accumulation of drug resistance mutations that may diminish the chances of viral resuppression with successive HAART regimens, and the long-term immune consequences of virologic rebound on HAART are not known. It is therefore important to evaluate the viral and immunologic responses among patients randomized to either an early or delayed HAART switch.

This study enrolls patients who have a viral load of at least 200, but less than 10,000 copies/ml. The patients are randomized into 2 treatment arms. Arm A (immediate switch) patients have genotypic resistance testing at entry. Based on the resistance test results, their antiretroviral treatment regimen is modified to a switch treatment regimen. Switch treatment initiates no later than Week 4. Arm B (delayed switch) patients continue their current antiretroviral regimen and have genotypic resistance testing when their plasma HIV-1 RNA levels reach 10,000 copies/ml or greater. Based on the resistance test results, their antiretroviral treatment regimen is modified to a switch treatment regimen. Switch treatment initiates no later than 4 weeks from the date of at least 10,000 copies/ml viral load, or from the date of an absolute CD4 count reduced by 20 percent from baseline value. Patients who never meet the switch criteria remain on study.

All patients are followed for a minimum of 48 weeks after entry. No antiretroviral drugs are provided by the study.

Recruitment & Eligibility

Status
COMPLETED
Sex
All
Target Recruitment
60
Inclusion Criteria

Not provided

Exclusion Criteria

Not provided

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Primary Outcome Measures
NameTimeMethod
Secondary Outcome Measures
NameTimeMethod

Trial Locations

Locations (23)

Harbor General/UCLA

🇺🇸

Torrance, California, United States

Univ of Miami School of Medicine

🇺🇸

Miami, Florida, United States

Northwestern Univ

🇺🇸

Chicago, Illinois, United States

Rush Presbyterian - Saint Luke's Med Ctr

🇺🇸

Chicago, Illinois, United States

The CORE Ctr

🇺🇸

Chicago, Illinois, United States

Harvard (Masschusetts General Hosp)

🇺🇸

Boston, Massachusetts, United States

Univ of Cincinnati

🇺🇸

Cincinnati, Ohio, United States

Univ of Pittsburgh

🇺🇸

Pittsburgh, Pennsylvania, United States

Univ of Texas, Southwestern Med Ctr of Dallas

🇺🇸

Dallas, Texas, United States

Univ of Texas Galveston

🇺🇸

Galveston, Texas, United States

Univ of Washington

🇺🇸

Seattle, Washington, United States

San Mateo AIDS Program / Stanford Univ

🇺🇸

Stanford, California, United States

Willow Clinic

🇺🇸

Menlo Park, California, United States

Miriam Hosp / Brown Univ

🇺🇸

Providence, Rhode Island, United States

Univ of Hawaii

🇺🇸

Honolulu, Hawaii, United States

Comprehensive Care Clinic / Vanderbilt Univ Med Ctr

🇺🇸

Nashville, Tennessee, United States

Univ of Colorado Health Sciences Ctr

🇺🇸

Denver, Colorado, United States

Brigham and Womens Hosp

🇺🇸

Boston, Massachusetts, United States

Stanford Univ Med Ctr

🇺🇸

Stanford, California, United States

Bellevue Hosp / New York Univ Med Ctr

🇺🇸

New York, New York, United States

Brown Univ / Miriam Hosp

🇺🇸

Providence, Rhode Island, United States

Univ of North Carolina

🇺🇸

Chapel Hill, North Carolina, United States

Duke Univ Med Ctr

🇺🇸

Durham, North Carolina, United States

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