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Efficacy of Spinal Cord Stimulation in Patients With Refractory Angina Pectoris

Not Applicable
Recruiting
Conditions
Refractory Angina Pectoris
Spinal Cord Stimulation
Interventions
Device: Spinal Cord Stimulator
Registration Number
NCT04915157
Lead Sponsor
Catharina Ziekenhuis Eindhoven
Brief Summary

There are a growing number of patients with refractory angina pectoris (RAP). RAP is defined as a 'chronic condition (\> three months) characterized by diffuse coronary artery disease in the presence of proven ischemia, which is not amendable to a combination of medical therapy, angioplasty or coronary bypass surgery'. These patients are severely restricted in performing daily activities due to debilitating angina complaints, leading to a decreased quality of life.

Spinal cord stimulation (SCS) is a last resort treatment option for patients with RAP. SCS is a device with a lead located in the thoracic epidural space and an Implantable Pulse Generator (IPG) in the abdomen or buttock that provides neurostimulation. Four possible mechanisms explaining the beneficial effects of SCS on RAP have been described: reduction of pain perception, decreased sympathetic tone, reduced myocardial oxygen demand, and improved coronary microcirculatory blood flow.

Research into the effect of SCS on RAP up to date have mainly been observational studies, with only four placebo-controlled randomized controlled trials. All studies confirm that treatment with SCS leads to a reduction in the number of angina pectoris attacks. What is currently not clear, is whether there is a placebo effect as results vary between the studies. One study looked at the effect of SCS in patients with RAP on the reduction of ischemia (using MIBI-SPECT) with no control arm. After 12 months myocardial ischemia was reduced, but not after three months of treatment. Leading to the conclusion that the reduction is myocardial ischemia was not a direct effect of SCS, but rather due to better coronary collateralization.

The 2020 ESC guideline 'chronic coronary syndromes' mentions non-existing to promising levels of evidence with regard to treatment options in patients with RAP and concludes that SCS may be considered (Class IIB; level of evidence B). It concludes that 'larger RCTs are required to define the role of each treatment modality for specific subgroups, to decrease non-responder rates and ascertain benefit beyond potential placebo effects'.

The aim of the current randomized controlled trial (double-blind, cross-over, placebo-controlled, single center) is to determine if high density spinal cord stimulation, a paresthesia free form of stimulation, leads to a significant reduction in myocardial ischemia (using PET with Rubidium-82 as tracer) in patients with refractory angina pectoris.

All patients included in this study will receive an implanted spinal cord stimulator after a positive TENS treadmill outcome and proven ischemia using the imaging modality PET with Rubidium-82 as tracer. Using a cross-over design all patients will have a 6 month period with high density stimulation and 6 month period of no stimulation. Randomization will determine in which order the patient receives these treatments. Both the patient and the treating physicians are blinded for this randomization process. At baseline a 6-minute walking test, the Seattle Angina Questionnaire, the RAND-36 questionnaire, the NRS scale and the CCS class will be performed/filled out.

Cross-over takes place at 6 months (switch from high density stimulation to no stimulation or vice versa) prior to which the PET scan is repeated, as well as the 6-minute walking test, the Seattle Angina Questionnaire, the RAND-36 questionnaire, the NRS-scale and the CCS-class.

At the end of the study period (12 months) the PET scan is repeated, as well as the 6-minute walking test, the Seattle Angina Questionnaire, the RAND-36 questionnaire, the NRS-scale and the CCS-class.

Detailed Description

Not available

Recruitment & Eligibility

Status
RECRUITING
Sex
All
Target Recruitment
72
Inclusion Criteria
  • Refractory Angina Pectoris:

    • Stable angina pectoris CCS class III or IV, with a minimum of 5 episodes of angina pectoris over the course of one week, during a minimum period of three months prior to screening
    • Coronary angiogram (CAG) performed within the last 12 months showing significant coronary artery disease defined as at least one coronary artery stenosis of >75% or 50 - 75% with proven ischaemia (see below), not suitable for revascularisation. Confirmed by one (or two in case of doubt) interventional cardiologist based on CAG images.
    • Optimal anti-anginal medication. Patients should at least use the maximal tolerable dose of a b-blocker, calcium channel blocker and short- and/or long-acting nitrate. If the patient doesn't use one of these groups of medication the reason (side-effects) should be clear.
  • Proven ischemia:

    • MIBI-SPECT: summed stress score (SSS) of at least 1, in combination with summed difference score (SDS) of at least 1 (1-4 mild ischaemia, > 4 moderate to severe ischaemia).
    • FFR: < 0.80, with no intervention options (determined by interventional cardiologist)
    • MRI perfusion: ≥ 1 segment of subendocardial hypoperfusion during stress perfusion, not present at rest and no matching fibrosis (using 16 segment AHA heart model)
    • PET: Semi-quantitative measurement: SSS score of at least 1, in combination with SDS score of at least 1 (1-4 mild ischaemia, > 4 moderate to severe ischaemia). Quantitative measurement: reduced myocardial perfusion reserve.
  • No revascularisation (PCI and/or CABG) performed between ischaemia testing and study inclusion.

  • Age > 18 years

Exclusion Criteria
  • Acute coronary syndrome (ACS) during the three month period prior to screening
  • Life expectancy less than 12 months
  • Inability to perform a 6-minute walking test
  • Inability to give informed consent
  • No proven ischemia
  • Spinal cord disease which could prevent correct positioning of the lead in the epidural space; to be determined by the anaesthesiologist performing the implantation
  • Anticoagulation therapy that cannot be stopped/bridged prior to spinal cord stimulator implantation
  • Inadequate paraesthesia coverage, during implantation, of the thoracic region where angina complaints are localized
  • Pregnancy
  • Mild Cognitive Impairment or dementia
  • Concomitant symptomatic valvular heart disease including severe aortic stenosis and/or regurgitation, severe mitral valve stenosis and/or regurgitation or severe tricuspid valve regurgitation.

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Group A: High Density stimulation - No StimulationSpinal Cord StimulatorPatients in this group will receive high density stimulation (parasthesia free form of stimulation) during the first 6 months of the study period. After 6 months cross-over will take place and patients will receive no stimulation during the final 6 months of the study period.
Group B: No Stimulation - High Density StimulationSpinal Cord StimulatorPatients in this group will receive no stimulation during the first 6 months of the study period. After 6 months cross-over will take place and patients will receive high density stimulation (parasthesia free form of stimulation) during the final 6 months of the study period.
Primary Outcome Measures
NameTimeMethod
Myocardial ischaemia6 months

The primary endpoint is the change in the percentage of myocardial ischaemia (% of left ventricular myocardium) measured using PET perfusion scan at the end of the six month period of HD stimulation compared to baseline.

Secondary Outcome Measures
NameTimeMethod
Grading of angina pectorisFrom date of randomization until six and twelve months later

Grading of angina pectoris using the Canadian Cardiovascular Society (CCS) class; grading scale of I - IV with higher scores representing worse outcome.

Frequency of angina pectoris attacksFrom date of randomization until six and twelve months later

Frequency of angina pectoris attacks measured using the Seattle Angina Questionnaire; scale 0 - 100 with higher scores representing a better outcome.

Patient conditionFrom date of randomization until six and twelve months later

Patient condition measured using the 6-minute walking test

Cardiovascular mortalityFrom date of randomization until six and twelve months later

Occurence of cardiovascular mortality

Severity of angina pectoris attacksFrom date of randomization until six and twelve months later

Severity of angina pectoris attacks using the Numeric Rating Scale (NRS-scale); scale 0 - 10 with higher scores representing a worse outcome.

Emergency room visits due to angina pectorisFrom date of randomization until six and twelve months later

Number of presentations at the emergency room due to angina pectoris

Changes in regional and global myocardial blood flow and myocardial flow reserveFrom date of randomization until six and twelve months later

Changes in the regional and global myocardial blood flow and myocardial flow reserve measured using PET perfusion scan

Hospital admissions due to acute coronary syndromeFrom date of randomization until six and twelve months later

Number of hospital admissions due to acute coronary syndrome (ACS)

Frequency of short-acting nitroglycerin useFrom date of randomization until six and twelve months later

Frequency of short-acting nitroglycerin use measured using the Seattle Angina Questionnaire; scale 0 - 100 with higher scores representing a better outcome.

RevascularizationFrom date of randomization until six and twelve months later

Occurence of revascularization (CABG and/or PCI)

Quality of life outcomeFrom date of randomization until six and twelve months later

Quality of life outcome measured using the RAND 36-Item Health Survery (RAND-36 questionnaire); scale 0 - 100 with higher scores representing a better outcome.

Trial Locations

Locations (1)

Catharina Hospital

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Eindhoven, Noord Brabant, Netherlands

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