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Clinical Trials/NCT04724668
NCT04724668
Completed
Not Applicable

The Role of the Circadian System in Binge Eating Disorder

University of Cincinnati1 site in 1 country43 target enrollmentJanuary 15, 2021

Overview

Phase
Not Applicable
Intervention
Not specified
Conditions
Binge-Eating Disorder
Sponsor
University of Cincinnati
Enrollment
43
Locations
1
Primary Endpoint
Phase 1 Dim Light Melatonin Onset (DLMO)
Status
Completed
Last Updated
4 months ago

Overview

Brief Summary

Binge eating disorder (BED) shows prominent circadian features that suggest a delay in circadian phase, and preliminary evidence shows binge eating may be responsive to chronobiological interventions, implicating a circadian system dysfunction in its pathophysiology. What remains lacking, however, is comprehensive knowledge of the characteristics of circadian system dysfunction in BED, and whether this dysfunction represents a therapeutic target in BED. There is therefore a critical need to characterize circadian system dysfunction in BED, and evaluate it as a potential therapeutic target. Without such information, the understanding on the role of the circadian system in BED and its potential as a new therapeutic target will remain limited.

Detailed Description

The overall objective of the research strategy will be to characterize circadian system dysfunction in BED and its potential as a therapeutic target. The central hypothesis is that a circadian system dysfunction (phase delay) plays a role in the pathophysiology of BED, and that advancing the circadian phase will improve BED symptoms. To attain the overall objectives, the following specific aims will be pursued in two phases: Specific aim 1) To characterize circadian system dysfunction in BED (Phase 1). Circadian system function will be evaluated in 80 adult (18 to 50yrs) obese subjects, 40 with BED and 40 without BED as a control group matched by age, body mass index (BMI), and gender, during a two-week observational phase. Based on preliminary data, the working hypothesis is that DLMO (the primary outcome measure) and secondary circadian parameters (i.e., locomotor activity acrophase) will occur later in the BED group compared with the control group, and a later circadian phase will be associated with worse BED clinical features. Specific aim 2) To evaluate circadian phase as a predictive biomarker for response to a chronobiological intervention and evidence of circadian system target engagement in BED (Phase 2). A mechanistic clinical trial with a 4-week double-blinded, randomized, sham/placebo controlled study design will evaluate the effect of a combination of morning lights+Melatonin/placebo on the circadian system and eating behavior on 40 BED subjects that complete phase 1. Subjects will be randomized to receive a combination of morning lights at usual wake time + Melatonin(3mg) or placebo (3hr before DLMO). Based on preliminary data, the working hypothesis is that a chronobiological intervention will induce a greater DLMO advance (primary outcome measure), greater decrease in binge eating days/week (secondary outcome measure), and change in exploratory metabolic outcomes. In addition, a later baseline DLMO (secondary outcome) will predict change in binge eating days/week and metabolic parameters in response to a chronobiological intervention.

Registry
clinicaltrials.gov
Start Date
January 15, 2021
End Date
May 30, 2025
Last Updated
4 months ago
Study Type
Interventional
Study Design
Parallel
Sex
All

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Francisco Romo-Nava

Associate Professor

University of Cincinnati

Eligibility Criteria

Inclusion Criteria

  • Not provided

Exclusion Criteria

  • Not provided

Outcomes

Primary Outcomes

Phase 1 Dim Light Melatonin Onset (DLMO)

Time Frame: Phase 1 baseline (visit 0)

Difference in mean DLMO (measured in time) between subjects with binge eating disorder (BED) and control subjects without BED.

Phase 2 Dim Light Melatonin Onset (DLMO)

Time Frame: Phase 2 baseline (visit 0) to endpoint, on average one month.

Differences in DLMO (measured in time) change from baseline to endpoint between two intervention groups will be analyzed using an ANCOVA model with age as a covariate.

Secondary Outcomes

  • Phase 1 Association between DLMO and binge eating days/week(Phase 1 baseline (visit 0))
  • Phase 2 Locomotor activity acrophase(Phase 2 baseline (visit 0) to endpoint, on average one month.)
  • Phase 1 Midline Estimating Statistic of Rhythm (MESOR)(Phase 1 baseline (visit 0))
  • Phase 2 baseline (visit 0) to endpoint(Phase 2 baseline (visit 0) to endpoint, on average one month.)
  • Phase 1 Locomotor activity acrophase(Phase 1 baseline (visit 0))
  • Phase 2 Binge eating days/week(Phase 2 baseline (visit 0) to endpoint, on average one month.)
  • Phase 1 MEQ(Phase 1 baseline (visit 0))

Study Sites (1)

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