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Clinical Trials/NCT02701348
NCT02701348
Completed
Not Applicable

Radiological and Biological Tumoural and Peri-tumoural Factors in Neoadjuvant Endocrine-treated Breast Cancers

NHS Tayside1 site in 1 country31 target enrollmentMay 2016
ConditionsBreast Cancer

Overview

Phase
Not Applicable
Intervention
Not specified
Conditions
Breast Cancer
Sponsor
NHS Tayside
Enrollment
31
Locations
1
Primary Endpoint
Change in Pathological response
Status
Completed
Last Updated
4 years ago

Overview

Brief Summary

The purpose of this study is to monitor the change in cancer size in women with breast cancer on anti-hormone treatment using different types of assessment including ultrasound scan (US), shearwave elastography (SWE) and magnetic resonance imaging (MRI), and assess how this corresponds to the changes in the cancer biology.

Detailed Description

Background Endocrine resistance is a significant problem in the management of breast cancer, with increasing evidence that the tumour microenvironment is influential on tumour growth and disease resistance. The neoadjuvant setting provides an excellent opportunity to observe tumour response to treatment in vivo, allowing development of methods for monitoring and predicting response to treatment. Aims To assess potential radiological and biological tumoural and peri-tumoural biomarkers in patients before and during neoadjuvant endocrine treatment. Our hypothesis is that there will be less response in women with abnormal peri-tumoural stroma, and that tumours with high monocarboxyl transporter (MCT4) and loss of caveolin-1 in stroma are resistant to endocrine treatment. Techniques and Methodology Patients with primary breast cancer receiving neoadjuvant letrozole will undergo radiological assessment with digital mammogram, US including SWE, and MRI. Core biopsies will be taken at diagnosis and at surgery from tumour and peri-tumoural stroma, and assessed for biomarkers lysyl oxidase (LOX), fibronectin, collagen, proliferation, MCT4 and caveolin-1. All data will be correlated to peri-tumoural abnormalities on MRI and SWE. Impact on breast cancer research This study will provide information on the ability of SWE and MRI to predict and detect endocrine resistance, correlated with biological markers that are associated with endocrine resistance. Identifying resistant tumours can prevent unnecessary treatment and reduce risks of recurrence as alternative or additional therapies can be utilised.

Registry
clinicaltrials.gov
Start Date
May 2016
End Date
December 24, 2020
Last Updated
4 years ago
Study Type
Interventional
Study Design
Single Group
Sex
Female

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Jane MacAskill

Dr. Jane MacAskill

NHS Tayside

Eligibility Criteria

Inclusion Criteria

  • Postmenopausal women: Defined as \>12 months amenorrhoea in absence of medical therapy known to induce this; or bilateral oophorectomy; or if last menses \<12 months before starting treatment, FSH \>35 IU/L and LH \>40 IU/L.
  • ER positive (Allred score \>3) invasive breast cancer
  • Staging as T1-4, N0-2, M0
  • Patient agreed to neoadjuvant endocrine therapy as recommended by MDT
  • Fresh tissue stored at time of diagnostic core biopsy
  • Suitable for, and tolerant of MRI scan
  • Fit for surgical intervention at time of entry into study

Exclusion Criteria

  • Premenopausal or unable to determine menopausal status
  • Not fit for surgical intervention due to co-morbidities
  • Contraindication for MRI (including severe claustrophobia)
  • Current use of HRT, or HRT use at time of diagnostic core biopsy

Outcomes

Primary Outcomes

Change in Pathological response

Time Frame: At surgery, min 3 months max 24 months

Residual cancer burden score

Change in Proliferative response

Time Frame: At surgery, min 3 months max 24 months

Pathological response to treatment by proliferation (Ki67) (%)

Change in Shearwave stiffness

Time Frame: 0,3,6, up to 24 months

shearwave stiffness (kPa)

Secondary Outcomes

  • Change in peritumoural imaging on MRI(0 months and surgery (min 3 months, max 24 months))
  • caveolin-1(0 months and at time of surgery (min 3 months, max 24 months))
  • Monocarboxyl transport 4(0 months and at time of surgery (min 3 months, max 24 months))
  • lysyl oxidase(0 months and at time of surgery (min 3 months, max 24 months))
  • fibronectin(0 months and at time of surgery (min 3 months, max 24 months))
  • collagen(0 months and at time of surgery (min 3 months, max 24 months))

Study Sites (1)

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