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Open-label Study of Neuraminidase Inhibitor Treatment in STEMI Patients

Phase 4
Conditions
STEMI
ST Elevation Myocardial Infarction
Interventions
Registration Number
NCT04684498
Lead Sponsor
Tongji Hospital
Brief Summary

Neuraminidase-1 can cause the removal of terminal sialic acid residues from the cell surface or serum sialyloconjugates. The level of Neu5Ac was positively related to the activity of neuraminidase-1. Elevation of Neu5Ac was observed in myocardial ischemia animal model, as well as patients with coronary artery disease. It is interesting to note that Neu5Ac and its regulatory enzyme neuraminidase-1 seem to play a key role in triggering myocardial ischemic injury. Oseltamivir, a structural mimic of sialic acid, was widely used as anti-influenza drug. It suppressed neuraminidase-1 activity in the heart. Targeting neuraminidase-1 may represent a new therapeutic intervention for coronary artery disease. This project seeks to identify whether neuraminidase inhibitor (Oseltamivir) treatment could decrease the myocardial infarct size in STEMI patients and improve clinical outcomes.

Detailed Description

Not available

Recruitment & Eligibility

Status
UNKNOWN
Sex
All
Target Recruitment
382
Inclusion Criteria
  1. Between the ages of 18 and 75, regardless of gender;
  2. STEMI should be diagnosed by two attending physicians or above, including history, clinical symptoms and signs;
  3. Participate voluntarily and sign informed consent, and can be followed up for more than one month.
Exclusion Criteria
  1. Allergic to oseltamivir;
  2. Creatinine clearance rate less than 60%;
  3. Severe liver insufficiency;
  4. Female patients who have or plan to become pregnant;
  5. Life expectancy less than one year;
  6. Patients refused to comply with the requirements of this study;
  7. According to the discretion of investigator, the patient was unable to complete the study or comply with the requirements of the study (for administrative or other reasons).

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Tamiflu (Oseltamivir Phosphate Capsules)Oseltamivir phosphate capsulesStandardized STEMI treatment + oseltamivir phosphate capsule (75mg, 2 times/day, 7days, oral)
no interventionOseltamivir phosphate capsulesStandardized STEMI treatment + no intervention
Primary Outcome Measures
NameTimeMethod
Myocardial infarct size at 1 week1 week

Myocardial infarct size at 1 week after acute myocardial infarction (quantified by Gadolinium-enhanced MRI).

Secondary Outcome Measures
NameTimeMethod
Myocardial infarct size based on culprit vessel with TIMI 0-1 blood flow1 week

Evaluated by coronary angiography and CMR.

Myocardial infarct size1 week

Myocardial infarct size under the curve of creatine kinase-MB (CK-MB) and hypersensitive troponin I.

The proportion of viable myocardium and ratio of myocardial reperfusion1 week

The proportion of viable myocardium and reperfusion was determined by the range of abnormal enhancement of gadolinium.

Composite end point at 1 week1 week

A composite end point of cardiogenic shock, cardiac death, malignant arrhythmia, and resuscitated cardiac arrest (including ventricular fibrillation) at 1 week.

Myocardial infarct size at 3 month3 month

Myocardial infarct size at 3 month after acute myocardial infarction (quantified by Gadolinium-enhanced MRI).

Composite end point at 6 month6 month

Composite end point at 6 month, including all-cause death, reinfarction, heart failure after myocardial infarction, and rehospitalization of unstable angina at 6 month.

Trial Locations

Locations (1)

Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology

🇨🇳

Wuhan, Hubei, China

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