MedPath

Ventricular Repolarization in Patients With Premature Ovarian Insufficiency (QT-IOP)

Not Applicable
Recruiting
Conditions
Premature Ovarian Insufficiency
Interventions
Diagnostic Test: ECG
Registration Number
NCT04167033
Lead Sponsor
Assistance Publique - Hôpitaux de Paris
Brief Summary

Ventricular repolarization, measured by corrected QT interval (QTc), is influenced by sex hormones. A QTc above 460msec predisposes to the risk of "torsades-de-pointes"(TdP). The investigators have recently shown that estradiol determines an increase in QTc elongation and progesterone shortens it. In addition, high gonadotropin levels (FSH or LH) are associated with QTc prolongation. Hypergonadotropic hypogonadisms (low progesterone and high gonadotropins) are therefore hormonal situations that promote QTc prolongation. Premature ovarian insufficiency (POI) is one of them. Its management is based on the prescription of hormone replacement therapy (HRT). Epidemiological studies have shown that these patients would be at increased risk of cardiovascular mortality. Our team is interested in the effect of this pathological hormonal situation and its HRT on ventricular repolarization in order to define whether this is a population at risk for long QTc.

Detailed Description

Ventricular repolarization, measured by the duration of the heart rate corrected QT interval (QTc), is influenced by sex hormones. A QTc above 460msec predisposes to the risk of torsades-de-pointes (TdP); ventricular arrhythmias that can lead to sudden death.

From puberty to menopause, QTc is longer in women than in men (\~10-15msec difference) and varies in women according to the menstrual cycle (\~5-10msec). This explains the increased risk of TdP in women compared to men. During the menstrual cycle, the risk is highest for women during the follicular phase compared to the luteal phase. The investigators have recently shown that estradiol determines an increase in QTc elongation and progesterone shortens it. In addition, high gonadotropin levels (FSH or LH) are associated with QTc prolongation. Hypergonadotropic hypogonadisms (low progesterone and high gonadotropins) are therefore hormonal situations that promote QTc prolongation.

Premature ovarian insufficiency (POI) affects 1% of women under 40 years of age and is characterized by hypergonadotropic hypogonadism. POI is associated with hormonal deficiencies responsible for amenorrhea and infertility. Management is based on the prescription of hormone replacement therapy (HRT). Epidemiological studies have shown that these patients would be at increased risk of cardiovascular mortality. HRT will be based on the combination of an estrogen and a progestin and will lead to a variable decrease in gonadotropins, depending on the steroid hormones/doses used. Our team, after structuring one of the largest international cohorts of patients with POI, is interested in the effect of this pathological hormonal situation and its HRT on ventricular repolarization to define whether this is a population at risk for long QTc. Indeed, ECG follow-up is recommended and many drugs (cardiovascular or not), are to be avoided, or even contraindicated in situations at risk of long QTc.

Recruitment & Eligibility

Status
RECRUITING
Sex
Female
Target Recruitment
120
Inclusion Criteria

Patients with POI

  • Patient aged 18 to 40 years
  • Patient with POI diagnostic criteria (FSH >25UI/l twice at intervals of a few weeks) with amenorrhea
  • No hormone treatment interacting with the gonadotropic axis for at least one month before inclusion
  • Patient who has signed informed consent
  • Patient affiliated to a social security system

Healthy volunteers (including POI control group)

  • Healthy women, aged 18 to 40 years, age-matched (+/- 5 years), and by BMI class (BMI<18, 18-25, 25-30, 30-35, 35-40, >40) compared to women with BPI
  • Women with regular cycles of 26 to 32 days
  • Women who has signed an informed consent form
  • Patient affiliated to a social security system
Exclusion Criteria

Patients with POI

  • Patient on HRT during the 1st evaluation
  • Pregnant or breastfeeding woman
  • Treatment regimen known to lengthen QT or act on ventricular repolarization
  • Cardiac history in particular cardiac rhythm disorder
  • Diabetes
  • Patient on AME (unless derogation from affiliation),
  • Severe renal insufficiency (MDRD <30ml/min/m²)

Healthy volunteers (including POI control group)

  • Diabetes or any chronic disease (including cardiovascular and endocrine)
  • Pregnant or breastfeeding woman
  • Hormonal contraceptive treatment in progress or stopped less than 3 months ago
  • Chronic treatment affecting the duration of QTc
  • Woman under AME (unless affiliation derogation)

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
healthy volunteersECG60 healthy volunteers matched with POI's patients
Primary Outcome Measures
NameTimeMethod
Duration of QTcin the luteal phase between Day22 and Day25

Compare the duration of QTc in patients with non-substituted POI with that of matched healthy volunteers on cardiovascular risk factors. The QTc will be measured by the Fridericia method

Secondary Outcome Measures
NameTimeMethod
Duration of QTcin the luteal phase between Day22 and Day25

Study the association between sex hormone levels (gonadotropins, steroid hormones) and QTc duration, as well as their variation in patients with POI and healthy volunteers

Trial Locations

Locations (3)

Hopital Haut Leveque

🇫🇷

Bordeaux, France

Pitié Salpêtrière

🇫🇷

Paris, France

BACHELOT

🇫🇷

Paris, France

© Copyright 2025. All Rights Reserved by MedPath