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Metabolic Adaptation to High-frequent Hypoglycaemia in Type 1 Diabetes

Not Applicable
Active, not recruiting
Conditions
Type1diabetes
Interventions
Procedure: Muscle biopsy
Procedure: Adipose tissue biopsy
Device: IPRO 2 Medtronic Minimed
Procedure: 7 Tesla (7T) Magnetic Resonance Imaging
Procedure: Indirect Calorimetry using Jaeger Oxycon Champion
Procedure: Core temperature and thermography using Thermovision SC645
Device: Freestyle Libre 2
Registration Number
NCT05095259
Lead Sponsor
Nordsjaellands Hospital
Brief Summary

An experimental mechanistic study. The overall objective is to gain new knowledge about mechanisms involved in adaptation to recurrent hypoglycaemia in diabetes by investigating patients with type 1 diabetes and healthy controls. The knowledge to be obtained may feed into experimental hypoglycaemic clamp studies to further elucidate the effect of the adaptations during acute hypoglycaemia. Ultimately, it may lead to intervention studies aiming at the maintenance of functional capability during hypoglycaemia in patients with type 1 diabetes to reduce their risk of severe hypoglycaemia.

Detailed Description

Study rationale The risk of severe hypoglycaemia is a major daily concern for people with diabetes treated with insulin. Severe hypoglycaemia is the main barrier in achieving the recommended glycaemic targets and may indirectly be the main driver for late diabetic complications and related morbidity, mortality and health care costs. In people with diabetes, recurrent exposure to insulin-induced mild hypoglycaemia leads to significant adaptive physiologic responses. While the metabolism of the brain and hormonal responses to hypoglycaemia have been studied extensively, this study will as the first, systematically investigate the chronic adaptation of peripheral metabolism to recurrent hypoglycaemia in diabetes. Knowledge about such responses can lead to interventions that attenuate the devastating effects of acute hypoglycaemia induced by insulin in people with diabetes. Thereby, the risk of developing severe hypoglycaemia can be reduced which ultimately will improve long-term diabetes outcomes and reduce health care costs.

Hypothesis Patients with type 1 diabetes that are exposed to high-frequent recurrent hypoglycaemia will adapt their metabolism in a way, which supports the preservation of brain fuelling.

Objectives

Primary objective The overall objective is to gain new knowledge about mechanisms involved in adaptation to recurrent hypoglycaemia in diabetes by investigating patients with type 1 diabetes and healthy controls. The knowledge to be obtained may feed into experimental hypoglycaemic clamp studies to further elucidate the effect of the adaptations during acute hypoglycaemia. Ultimately, it can lead to intervention studies aiming at the maintenance of functional capability during hypoglycaemia in patients with type 1 diabetes to reduce their risk of severe hypoglycaemia.

Secondary objectives

* To study the metabolic consequences of recurrent hypoglycaemia in the brain, liver, muscle and adipose tissues

* To study the consequences of recurrent hypoglycaemia on resting metabolic rest

* To study the consequences of recurrent hypoglycaemia on glucagon and adrenaline sensitivity

* To study the consequences of recurrent hypoglycaemia on epigenetic profiles

* To study the consequences of recurrent hypoglycaemia on oxidative stress

* To study the psychological factors associated with recurrent hypoglycaemia

Recruitment & Eligibility

Status
ACTIVE_NOT_RECRUITING
Sex
All
Target Recruitment
60
Inclusion Criteria
  1. Ability to provide written informed consent
  2. Male or female aged 18-70 years
  3. Must be able to speak and read Danish
  4. Type 1 diabetes patients or healthy individuals (control goup)
  5. A documented clinically relevant history of type 1 diabetes
  6. In insulin treatment regimen
  7. The subject must be willing and able to comply with trial protocol
Exclusion Criteria
  1. History of severe psychological condition
  2. History of severe heart disease
  3. History of epilepsy, former apoplexies and dementia
  4. History of muscle diseases
  5. History of liver disease
  6. History of malignancy unless a disease-free period exceeding 5 years
  7. Implants not compatible for MRI scans
  8. History of alcohol or drug abuse
  9. Pregnant or lactating woman

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Participants with Type 1 Diabetes MellitusAdipose tissue biopsyParticipants with Type 1 Diabetes Mellitus
Participants with Type 1 Diabetes MellitusIndirect Calorimetry using Jaeger Oxycon ChampionParticipants with Type 1 Diabetes Mellitus
Participants with Type 1 Diabetes MellitusCore temperature and thermography using Thermovision SC645Participants with Type 1 Diabetes Mellitus
Participants with Type 1 Diabetes MellitusFreestyle Libre 2Participants with Type 1 Diabetes Mellitus
Healthy ControlsMuscle biopsyHealthy Controls
Healthy ControlsGlucagonHealthy Controls
Participants with Type 1 Diabetes MellitusIPRO 2 Medtronic MinimedParticipants with Type 1 Diabetes Mellitus
Healthy ControlsAdipose tissue biopsyHealthy Controls
Healthy ControlsIndirect Calorimetry using Jaeger Oxycon ChampionHealthy Controls
Participants with Type 1 Diabetes MellitusMuscle biopsyParticipants with Type 1 Diabetes Mellitus
Participants with Type 1 Diabetes Mellitus7 Tesla (7T) Magnetic Resonance ImagingParticipants with Type 1 Diabetes Mellitus
Healthy ControlsIPRO 2 Medtronic MinimedHealthy Controls
Healthy ControlsCore temperature and thermography using Thermovision SC645Healthy Controls
Healthy Controls7 Tesla (7T) Magnetic Resonance ImagingHealthy Controls
Participants with Type 1 Diabetes Mellitusinsulin humanParticipants with Type 1 Diabetes Mellitus
Participants with Type 1 Diabetes MellitusEpinephrinParticipants with Type 1 Diabetes Mellitus
Participants with Type 1 Diabetes MellitusGlucagonParticipants with Type 1 Diabetes Mellitus
Healthy ControlsEpinephrinHealthy Controls
Healthy Controlsinsulin humanHealthy Controls
Primary Outcome Measures
NameTimeMethod
Glycogen in muscle and adipose tissue5 minutes

Glycogen in muscle and adipose tissue biopsies during euglycaemia

Brain adenosine triphosphate (ATP) concentration20 minutes

Brain ATP concentration using non-invasive MR spectroscopy during euglycaemia

Glycogen concentration40 minutes

Glycogen in liver and muscle tissue using non-invasive MR spectroscopy during euglycaemia.

Metabolite- and lipid profiling5 minutes

Metabolite- and lipid profiling of blood samples using metabolomics profiling platforms during euglycaemia

Brain lactate concentration20 minutes

Brain lactate concentration using non-invasive magnetic resonance (MR) spectroscopy during euglycaemia

Non-specific proteins in muscle and adipose tissue5 minutes

Non-specific proteins in muscle and adipose tissue biopsies during euglycaemia

Secondary Outcome Measures
NameTimeMethod
Plasma β-hydroxybutyrate during glucagon injections.Every 40 minutes up to 5 hours

Plasma β-hydroxybutyrate during glucagon injections.

Plasma metabolomics during glucagon injections.Every 40 minutes up to 5 hours

Plasma metabolomics during glucagon injections.

Plasma alanine during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma alanine during epinephrine infusion

Plasma β-hydroxybutyrate during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma β-hydroxybutyrate during epinephrine infusion

Plasma insulin during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma insulin during epinephrine infusion

Plasma norepinephrine during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma catecholamines during epinephrine infusion

Plasma metabolomics during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma metabolomics during epinephrine infusion

Personality traits using the psychometry questionnaire Toronto Alexithymia Scale (TAS-20)30 minutes

Personality traits using the psychometry questionnaire TAS-20, score 20-100, the higher score the more likely they are alexithymia

Diabetes and hypoglycaemia status using psychometry questionnaire Hypoglycemia Fear Survey - Worry (HFS-W)30 minutes

Diabetes and hypoglycaemia status using psychometry questionnaire HFS-W, score 0-72, the higher score the higher fear for hypoglycemia

Estimated glucose production during glucagon stimulationEvery 5 minutes up to 5 hours

Area under the curve (AUC) for plasma glucose during glucagon injections. Plasma glucose measurement

Plasma lactate during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma lactate during epinephrine infusion

Plasma epinephrine during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma catecholamines during epinephrine infusion

Personality traits using the psychometry questionnaire Type D Scale-14 (DS-14)30 minutes

Personality traits using the psychometry questionnaire DS-14, score between 0-28, the higher, the more likely they have type D personality

Diabetes and hypoglycaemia status using psychometry questionnaire Problem Areas in Diabetes (PAID)30 minutes

Diabetes and hypoglycaemia status using psychometry questionnaire PAID, 0-80, the higher score, the more problems with diabetes

Food consumption30 minutes

Using Food Frequency Questionnaire to analyze food consumption

Hypoglycemia awareness status10 minutes

Using hypoglycemia awareness status questionnaire

, 0-7, higher score indicate hypoglycemia unawareness

Indirect calorimetry60 minutes

Estimating resting metabolic rate, before and during hyperinsulinemic-hypoglycemic clamp

Plasma lactate during glucagon injections.Every 40 minutes up to 5 hours

Plasma lactate during glucagon injections.

Plasma free fatty acids during glucagon injections.Every 40 minutes up to 5 hours

Plasma free fatty acids during glucagon injections.

Plasma glycerol during glucagon injections.Every 40 minutes up to 5 hours

Plasma glycerol during glucagon injections.

Plasma alanine during glucagon injections.Every 40 minutes up to 5 hours

Plasma alanine during glucagon injections.

Plasma insulin during glucagon injections.Every 40 minutes up to 5 hours

Plasma insulin during glucagon injections.

Plasma glycerol during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma glycerol during epinephrine infusion

Plasma glucagon during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma glucagon during epinephrine infusion

Estimated glucose production during epinephrine stimulationEvery 5 minutes up to 90 minutes

Area under the curve (AUC) for plasma glucose during epinephrine infusion. Plasma glucose measurement

Thermography5 minutes

Estimating skin temperature, before and during hyperinsulinemic-hypoglycemic clamp

Diabetes and hypoglycaemia status using psychometry questionnaire Hypoglycemia Attitudes and Behavior Scale (HABS)30 minutes

Diabetes and hypoglycaemia status using psychometry questionnaire HABS, score from 14-45, higher score more fear of hypoglycemia

Plasma glucagon during glucagon injections.Every 40 minutes up to 5 hours

Plasma glucagon during glucagon injections.

Plasma free fatty acids during epinephrine infusionEvery 20 minutes up to 90 minutes

Plasma free fatty acids during epinephrine infusion

Trial Locations

Locations (2)

Nordsjaellands Hospital

🇩🇰

Hillerød, Denmark

Steno Diabetes Center Copenhagen

🇩🇰

Gentofte, Denmark

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