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nderstanding how treatments that reduce self-attacking immune proteins affect gene readouts in ANCA-associated vasculitis

Not Applicable
Completed
Conditions
nderstanding of the mechanism of B cell depletion in ANCA-associated vasculitis
Circulatory System
Registration Number
ISRCTN16128829
Lead Sponsor
Cambridge University Hospitals NHS Foundation Trust
Brief Summary

Not available

Detailed Description

Not available

Recruitment & Eligibility

Status
Completed
Sex
All
Target Recruitment
188
Inclusion Criteria

Participants from the RITAZAREM clinical trial who have provided biological samples as part of the original study protocol

Exclusion Criteria

Patients who were not enrolled in the RITAZAREM clinical trial

Study & Design

Study Type
Observational
Study Design
Not specified
Primary Outcome Measures
NameTimeMethod
Transcriptomic analysis of peripheral blood samples using bulk RNA sequencing at baseline<br>and months 4, month 24, and month 36
Secondary Outcome Measures
NameTimeMethod
1. Immune cell proportions following B cell depletion therapy, identified using deconvolution analysis at baseline and months 4, month 24, and month 36<br>2. Cell-intrinsic immune cell transcriptomes following B cell depletion, identified using deconvolution analysis at baseline and months 4, month 24, and month 36<br>3. Immune cell proportions and transcriptomes in rituximab and azathioprine-treated individuals, identified using deconvolution analysis at baseline and months 4, month 24, and month 36<br>4. Previously identified T cell exhaustion signatures known to be associated with clinical outcomes in AAV, assessed using deconvolution analysis at baseline and months 4, month 24, and month 36<br>5. Cell proportions or cell-intrinsic transcriptomes after withdrawal of immunosuppression in those who relapse against those who do not, identified using deconvolution analysis at baseline and months 4, month 24, and month 36
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