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Clinical Trials/2024-514494-21-00
2024-514494-21-00
Recruiting
Phase 2

A Multicenter Randomized, Controlled, Double-blinded Trial to Evaluate Efficacy and Safety of Bortezomib in Patients With Severe Autoimmune Encephalitis

Friedrich-Schiller-Universitaet Jena17 sites in 1 country50 target enrollmentMay 13, 2020

Overview

Phase
Phase 2
Intervention
Not specified
Conditions
Not specified
Sponsor
Friedrich-Schiller-Universitaet Jena
Enrollment
50
Locations
17
Primary Endpoint
mRS 17 weeks after first administration of the investigational product
Status
Recruiting
Last Updated
8 months ago

Overview

Brief Summary

Evaluation of the efficacy and safety of bortezomib in patients with severe autoantibody-positive autoimmune encephalitis

Detailed Description

Autoimmune encephalitis is characterized by autoantibodies against neuronal surface antigens like the NMDA (N-methyl-D-aspartate) receptor or LGI1 (Leucin-rich glioma inactivated protein 1). So far, no specific therapy exists for this disease. Actual treatment includes combination therapies aiming for a reduction of pathogenic antibodies and containing the autoimmune process. In first line, patients are treated with plasmapheresis and cortisone. In second line, Rituximab and/or cyclophosphamide are administered. The response to these treatments are, however, often delayed and insufficient. Therefore, we need a specific therapy aiming at the antibody-producing plasma cells. Bortezomib is a proteasome inhibitor which interferes with NF-kB (nuclear factor kB) and the ubiquitin proteasome signaling pathway. Bortezomib acts preferably on cells with high protein synthesis - like plasma cells - and induces cell death in these cells. Bortezomib is used since more than a decade in chemotherapy of the multiple myeloma. Additionally, it is reported for systemic autoimmune diseases like lupus erythematodes that bortezomib leads to a depletion of plasma cells and therefore reduces the number of pathogenic antibodies and improves clinical outcome. The therapeutic potential of bortezomib for NMDAR encephalitis is described in a first case series with 5 patients.

Registry
euclinicaltrials.eu
Start Date
May 13, 2020
End Date
December 31, 2026
Last Updated
8 months ago
Study Type
Interventional
Study Design
Parallel

Investigators

Responsible Party
Principal Investigator
Principal Investigator

Prof. Dr. Christian Geis

Scientific

Friedrich-Schiller-Universitaet Jena

Eligibility Criteria

Inclusion Criteria

  • Clinically diagnosed severe autoimmune encephalitis (defined as mRS ≥ 3)
  • Autoantibodies against neuronal surface proteins in cerebrospinal fluid or serum serum, detection must not be older than 4 weeks, calculated before randomization
  • Pre-treatment with rituximab
  • Age ≥ 18 years
  • Written informed consent of the patient or the patient “under witness” (if the patient cannot write for motor reasons) cannot write themselves) or the legal representative (=guardian) or the authorized representative
  • Potentially fertile patient (up to 2 years after menopause): negative pregnancy test

Exclusion Criteria

  • Acute infiltrative lung disease
  • Acute infiltrative pericardial disease
  • Malignant tumor under ongoing or newly started chemotherapy
  • Concurrent participation in another intervention study
  • Previous participation in this study
  • Known hypersensitivity to any ingredient of the investigational product
  • Continued therapy with glucocorticoids/rituximab during the duration of the study (last administration must be completed before first administration of investigational product)

Outcomes

Primary Outcomes

mRS 17 weeks after first administration of the investigational product

mRS 17 weeks after first administration of the investigational product

Secondary Outcomes

  • mRS and GCS 3, 6, 9 and 13 weeks after first administration of the investigational product; GCS 17 weeks after first administration of the investigational product
  • Length of stay in hospital/intensive care unit
  • Antibody titers and destruction markers (in serum and cerebrospinal fluid), cellular immune response (FACS, in cerebrospinal fluid) at the baseline visit and 17 weeks after first administration of the investigational product
  • Neurocognitive function (MoCA, MMST, VLMT and NPI) at the baseline visit and 17 weeks after the first visit and 17 weeks after first administration of the investigational product
  • Number of all (serious) adverse events within 17 weeks after the first 17 weeks after first administration of the investigational product
  • Bortezomib safety with regard to polyneuropathy, increase in liver enzymes liver enzymes, hematotoxicity, gastrointestinal toxicity and secondary infections.

Study Sites (17)

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