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Clinical Trials/NCT05280600
NCT05280600
Recruiting
Not Applicable

Developing Advanced Neuroimaging for Clinical Evaluation of Autoimmune Encephalitis

King's College London1 site in 1 country75 target enrollmentMay 19, 2022

Overview

Phase
Not Applicable
Intervention
Not specified
Conditions
Anti-N-Methyl-D-Aspartate Receptor Encephalitis
Sponsor
King's College London
Enrollment
75
Locations
1
Primary Endpoint
Structural MRI
Status
Recruiting
Last Updated
2 years ago

Overview

Brief Summary

Autoimmune encephalitis is brain inflammation caused by the immune system mistakenly reacting against proteins in the brain. The commonest form is called NMDAR-antibody encephalitis (N-methyl-D-aspartate receptor antibody encephalitis), a rare condition which mainly affects children and young people and causes difficulties in memory, thinking and mental health which can have significant long-term impacts on education, employment and quality of life.

In this project we will use advanced magnetic resonance imaging (MRI) to measure changes in the structure, function and chemistry of the brains of children and young people who are in early recovery from NMDAR-antibody encephalitis and other forms of immune-mediated encephalitis. We will investigate if MRI measurements in patients differ from those in healthy people, and if they can help predict patient outcome one year later, assessed by tests of memory, thinking, mental health and functioning in daily life.

Detailed Description

This study aims to develop non-invasive, in vivo measures of neurobiological dysfunction derived from the overarching hypothesis that dysfunction of inhibitory interneurons alters the cerebral concentrations of gamma-aminobutyric acid (GABA) and glutamate (Glu) and underlies T2 changes and deficient connectivity in functional networks in early recovery from NMDAR-antibody encephalitis. Our ambition is to identify the best potential prognostic biomarkers from these neurometabolite measurements and structural and functional MRI. Our primary objective is to test the following specific hypotheses in children and young people with NMDAR-antibody encephalitis: * Hypothesis 1: GABA is decreased, and Glu increased, on MR spectroscopy of the medial temporal lobe and medial prefrontal cortex in NMDAR-antibody encephalitis. * Hypothesis 2: Local GABA and Glu are correlated with (i) resting-state functional MRI (fMRI) based functional connectivity and (ii) parameter map-based microstructural changes. Specifically, we hypothesise that (i) GABA is positively correlated and Glu inversely correlated with functional connectivity, assessed by whole-brain mapping of the default mode network and seed-based analysis of hippocampal-frontal connectivity; and (ii) Glu is positively correlated and GABA inversely correlated with median T2 values within the hippocampus. * Hypothesis 3: Local neurometabolites, network measures and microstructural changes predict cognitive, psychiatric and functional outcome at one year. Specifically, we hypothesise that medial temporal Glu, GABA and hippocampal T2 predict memory performance, and prefrontal Glu and GABA predict attention, executive function and fluid intelligence.

Registry
clinicaltrials.gov
Start Date
May 19, 2022
End Date
February 28, 2026
Last Updated
2 years ago
Study Type
Observational
Sex
All

Investigators

Responsible Party
Sponsor

Eligibility Criteria

Inclusion Criteria

  • Not provided

Exclusion Criteria

  • Not provided

Outcomes

Primary Outcomes

Structural MRI

Time Frame: Baseline

Quantitative MRI parameter maps including measurement of median T2 values in the hippocampus

Resting-state fMRI

Time Frame: Baseline

Whole-brain mapping of the default mode network and seed-based analysis of hippocampal-frontal connectivity

Cerebral concentrations of GABA and glutamate at the prefrontal cortex and left medial temporal lobe

Time Frame: Baseline

Measured with MR spectroscopy - stimulated echo acquisition mode (STEAM) sequence

Secondary Outcomes

  • Wechsler Abbreviated Scale of Intelligence 2nd Edition (WASI-II)(Baseline (all groups), 1 year (patients))
  • CANTAB (Cambridge Cognition, UK): Paired Associates Learning(Baseline (all groups), 1 year (patients))
  • Prodromal Questionnaire Brief Version (PQ-B)(Baseline (all groups), 1 year (patients))
  • Patient Health Questionnaire (PHQ-9)(Baseline (all groups), 1 year (patients))
  • Generalized Anxiety Disorder 7-item Scale (GAD-7)(Baseline (all groups), 1 year (patients))
  • Pediatric Quality of Life Inventory (PedsQL) Generic Core Scales(Baseline (all groups), 1 year (patients))
  • CANTAB (Cambridge Cognition, UK): Intra-Extra Dimensional Set Shift(Baseline (all groups), 1 year (patients))
  • CANTAB (Cambridge Cognition, UK): Stockings of Cambridge(Baseline (all groups), 1 year (patients))
  • Rey Auditory Verbal Learning Test (RAVLT)(Baseline (all groups), 1 year (patients))
  • Doors & People Test(Baseline (all groups), 1 year (patients))
  • CANTAB (Cambridge Cognition, UK): Rapid Visual Information Processing(Baseline (all groups), 1 year (patients))
  • CANTAB (Cambridge Cognition, UK): Spatial Span(Baseline (all groups), 1 year (patients))
  • CANTAB (Cambridge Cognition, UK): Stop Signal Task(Baseline (all groups), 1 year (patients))
  • PedsQL Multidimensional Fatigue Scale(Baseline (all groups), 1 year (patients))
  • Behaviour Rating Inventory of Executive Function (BRIEF)(Baseline (all groups), 1 year (patients))
  • Conners 3 Short Form / Conners' Adult ADHD Rating Scale(Baseline (all groups), 1 year (patients))

Study Sites (1)

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