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Clinical Trials/NCT01377467
NCT01377467
Completed
Phase 3

A Phase 3, Investigator-initiated, Randomized, Open-label Single-center Study of the Effect of Denosumab on the Prevention of Bone Mineral Density Loss After Renal Transplantation

Rudolf Wuethrich1 site in 1 country90 target enrollmentStarted: June 2011Last updated:

Overview

Phase
Phase 3
Status
Completed
Sponsor
Rudolf Wuethrich
Enrollment
90
Locations
1
Primary Endpoint
Percent Change in BMD at the Total Lumbar Spine From Baseline to Month 12

Overview

Brief Summary

The primary objective of the study is to examine the effect of denosumab on lumbar spine bone mineral density (BMD) after one year of treatment in newly transplanted renal allograft recipients. Secondary endpoints include BMD changes at the total hip and the femoral neck, changes in body height, changes in bone mineral metabolism parameters, incidence of fractures, and allograft function at one year. Safety measurements include the occurrence of rejection episodes, infectious complications, graft loss and mortality.

  • Trial with medicinal product

Detailed Description

Renal allograft recipients are at high risk to suffer a substantial loss of bone mineral density (BMD) within the first year after kidney transplantation. This loss of BMD correlates with an increased risk for the development of osteoporosis or worsening of pre-existing osteopenia/osteoporosis, heightening the risk for the subsequent occurrence of fractures. Renal allograft recipients are often treated with calcium and vitamin D preparations to prevent BMD loss. The addition of bisphosphonates can further improve BMD. However, bisphosphonates are potentially nephrotoxic and promote adynamic bone disease, and are therefore not regularly prescribed.

Receptor Activator of Nuclear factor- Kappa-B Ligand (RANKL) is a key molecule mediating development, activity, and survival of osteoclasts. Osteoporosis results in part from increased osteoclastic bone resorption, and therefore the inhibition of RANKL activity has become an obvious therapeutic strategy to prevent bone mineral density (BMD) loss and the development of osteoporosis.

The novel anti-osteoporotic drug denosumab (trade name Prolia®) is a fully human monoclonal antibody against RANKL. By inhibiting the development and the activity as well as reducing the survival of osteoclasts it decreases bone resorption and increases bone density.

The hypothesis of the present study is that denosumab has a beneficial effect on the loss of BMD in the first year after renal transplantation. The preservation of BMD is a surrogate parameter, generally predicting subsequent improvements in the occurrence rate of fractures. The hypothesis will be tested by studying the effect of denosumab on BMD in newly transplanted renal allograft recipients.

The purpose of the present trial is to study the effect of denosumab on BMD in kidney allograft recipients. The study participants will be treated for 1 year, receiving a total of 2 injections of the standard 60 mg dose at baseline and at 6 months.

Ninety sequential renal allograft recipients will be randomized 1:1 to receive two subcutaneous 60 mg denosumab injections within 14 days and 6 months following renal transplantation, or no treatment. All patients will also receive oral standard treatment with 1000 mg calcium plus 800 IU vitamin D.

Study Design

Study Type
Interventional
Allocation
Randomized
Intervention Model
Parallel
Primary Purpose
Treatment
Masking
None

Eligibility Criteria

Ages
18 Years to 80 Years (Adult, Older Adult)
Sex
All
Accepts Healthy Volunteers
No

Inclusion Criteria

  • Not provided

Exclusion Criteria

  • Not provided

Arms & Interventions

Denosumab

Experimental

60 mg denosumab s.c. at baseline and after 6 months

Intervention: Denosumab (Prolia) (Drug)

Outcomes

Primary Outcomes

Percent Change in BMD at the Total Lumbar Spine From Baseline to Month 12

Time Frame: Baseline and month 12

The total lumbar spine BMD was measured via Dual Energy X-ray Absorptiometry (DXA) and was expressed in g/cm2 hydroxylapatite

Secondary Outcomes

  • Percent Change in BMD at the Total Hip From Baseline to Month 12(Baseline and month 12)
  • Percent Change in BMD at the Femoral Neck From Baseline to Month 12(Baseline and month 12)
  • Percent Change in BMD at the Total Lumbar Spine From Baseline to Month 6(Baseline and month 6)
  • Percent Change in BMD at the Total Hip From Baseline to Month 6(Baseline and month 6)
  • Percent Change in BMD at the Femoral Neck From Baseline to Month 6(Baseline and month 6)
  • Beta-CTX at Baseline and Months 3, 6 and 12(baseline, month 3, month 6, and month 12)
  • P1NP at Baseline and Months 3, 6 and 12(baseline, month 3, month 6, and month 12)

Investigators

Sponsor
Rudolf Wuethrich
Sponsor Class
Other
Responsible Party
Sponsor Investigator
Principal Investigator

Rudolf Wuethrich

Professor and Director

University of Zurich

Study Sites (1)

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