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Clinical Trials/NCT03325023
NCT03325023
Unknown
Phase 4

Prospective, Randomized, Double - Blind, Placebo Controlled Trial of Dietary Modification in Conjunction With Probiotic Therapy on Clinical and Endocrinological Parameters as Well as Body Composition in Women With Polycystic Ovary Syndrome

Poznan University of Medical Sciences1 site in 1 country80 target enrollmentMay 20, 2017
ConditionsPCOS

Overview

Phase
Phase 4
Intervention
Not specified
Conditions
PCOS
Sponsor
Poznan University of Medical Sciences
Enrollment
80
Locations
1
Primary Endpoint
Reduction of body mass and body fat percentage
Last Updated
8 years ago

Overview

Brief Summary

Polycystic ovary syndrome (PCOS) is the most common endocrinopathy affecting women of reproductive age. The pathogenesis of PCOS is not fully understood. The intestinal microbiota are believed to be associated with the development of insulin resistance and obesity, and therefore contributing to the development of PCOS. Incresed permeability of the intestinal mucosal barier and absorbtion of lipoproteinase (LPS) from G (-) bacteria promotes chronic inflammation and may lead to insulin resistance.

Approximately 50-60% of women suffering from PCOS are obese. It is known that lifestyle modification and body mass reduction improves endocrine parameters and restores ovulatory menstrual cycles in most patients. Currently, the use of probiotics and prebiotics is playing an increasingly important role in the treatment of obesity through the modulation of intestinal microflora.

The objectives of the study are based on the following assumptions:

  1. Insulin resistance and compensatory hyperinsulinemia are important aspects in the pathogenesis of PCOS and co-morbidity of cardiovascular disease.
  2. Aberrations in the intestinal microflora are associated with the development of obesity and insulin resistance.
  3. Dietary modification combined with probiotic supplementation improves endocrine and metabolic profiles in women with PCOS.

Detailed Description

Polycystic ovary syndrome (PCOS) is the most common endocrinopathy affecting women of reproductive age. The pathogenesis of PCOS is not fully understood. The intestinal microbiota are believed to be associated with the development of insulin resistance and obesity, and therefore contributing to the development of PCOS. Incresed permeability of the intestinal mucosal barier and absorbtion of lipoproteinase (LPS) from G (-) bacteria promotes chronic inflammation and may lead to insulin resistance. The microbiome consists of a complex community of microorganisms that live in the digestive tract. Intestinal microflora is colonized by various microorganisms, with the largest groups consisting of the Firmicutes, Bacteroides, Actinobacteria and Proteobacteria. Intestinal microflora has a beneficial effect on the host by competing with pathogenic bacteria, protecting the inegrity of the intestinal mucosal barier and stimulating the immune system. Some human gut microorganisms are involved In fermenting dietary fiber into short-chain fatty acids (SCFAs), such as acetic acid and butyric acid, which are then absorbed by the host. The most beneficial species of microbiota are Lactobacillus and Bifidobacterium. Approximately 50-60% of women suffering from PCOS are obese. It is known that lifestyle modification and body mass reduction improves endocrine parameters and restores ovulatory menstrual cycles in most patients. Currently, the use of probiotics and prebiotics is playing an increasingly important role in the treatment of obesity through the modulation of intestinal microflora. The objectives of the study are based on the following assumptions: 1. Insulin resistance and compensatory hyperinsulinemia are important aspects in the pathogenesis of PCOS and co-morbidity of cardiovascular disease. 2. Aberrations in the intestinal microflora are associated with the development of obesity and insulin resistance. 3. Dietary modification combined with probiotic supplementation improves endocrine and metabolic profiles in women with PCOS.

Registry
clinicaltrials.gov
Start Date
May 20, 2017
End Date
October 20, 2018
Last Updated
8 years ago
Study Type
Interventional
Study Design
Parallel
Sex
Female

Investigators

Sponsor
Poznan University of Medical Sciences
Responsible Party
Principal Investigator
Principal Investigator

Beata Banaszewska

MD PhD Associate Proffesor Division of Infertility and Reproductive Endocrinology, Department of Gynecology and Obsterics

Poznan University of Medical Sciences

Eligibility Criteria

Inclusion Criteria

  • Written consent for participation in the clinical trial
  • Age 18 to 45 years Irregular menstruation (\> 35 days) or secondary amenorrhea\> 3 months
  • Hyperandrogenism (hirsutism and / or acne) and / or hyperandrogenemia (total serum testosterone\> 0.5 ng / mL)

Exclusion Criteria

  • Ovarian cancer, adrenal gland tumor, endometrial cancer, cervical cancer, breast cancer
  • Congenital adrenal hyperplasia (17-OH-progesterone\> 2.5 ng / mL)
  • Clinically diagnosed Cushing's disease, acromegaly, gigantism
  • Type I or II diabetes
  • Unexplained bleeding from the genital tract
  • Hormone treatment within the last 2 months

Outcomes

Primary Outcomes

Reduction of body mass and body fat percentage

Time Frame: 6 months

Assessed using the Tanita MC-980 Body Composition Analyzer

Secondary Outcomes

  • Decrease of testosterone levels(6 months)
  • Increased regularity of menstrual cycle(6 months)
  • Improved homeostasis of the intestinal microflora(6 months)

Study Sites (1)

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