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Evaluation of Endothelial Dysfunction Using the "Flow Mediated Dilation" Test in a Population of Chronic Renal Failure Patients at Different Stages, and Evaluation of the Role of Antiphospholipid Antibodies

Not Applicable
Recruiting
Conditions
Chronic Renal Failure
Interventions
Procedure: Urine sampling
Procedure: Blood sampling
Device: Flow mediated dilatation test
Registration Number
NCT06347029
Lead Sponsor
Brugmann University Hospital
Brief Summary

The vascular endothelium is an organ in its own right, playing, among other things, a primordial role in the control of vascular tone. This vascular tone is ensured by pro-dilator mediators (nitric oxide (NO), prostacyclins (PGI2)), or vasoconstrictors (endothelin, thromboxane A2 or PGH2).Uremic toxin accumulation in chronic kidney disease (CKD) is a well-known factor in endothelial dysfunction, often associated with higher cardiovascular risk. This association is also present for terminal chronic kidney disease characterized by the need to resort to an extra-renal purification technique (in-center hemodialysis (HD), daily home hemodialysis (HDQ), peritoneal dialysis) or to resort to renal transplantation.

For HD to be effective, it is essential that the blood flow rate passing through the dialyzer is greater than 300ml/min. This imperative requires that any hemodialysis patient have specific vascular access (dialysis catheter or arteriovenous fistula (AVF)) to ensure these flow rates. The vascular access of choice is the arteriovenous fistula , because it is associated with a lower risk of infection and lower morbidity and mortality. Making an arteriovenous fistula consists of surgically creating an anastomosis between a vein and an artery.

Complications of arteriovenous fistula are common. Arteriovenous fistula maturation may be delayed (maturation delay) or even absent. Drainage veins and/or anastomoses can also be the site of stenosis or thrombosis. The pathophysiology of these complications is complex and multifactorial. Among the risk factors for these complications (delay or absence of maturation, stenosis thrombosis), the positivity of antiphospholipid antibodies (aPL) can be cited, as well as endothelial dysfunction.

Antiphospholipid syndrome (APS) is an autoimmune disease causing a thrombotic phenotype. This is an acquired thrombophilia. In the general population, the prevalence of antiphospholipid antibodies is around 0.5%; this prevalence is far from rare in hemodialysis, since it represents up to 37% in dialysis patients. In a retrospective study carried out at Brugmann University Hospital in 2023 , on 115 patients with AVF and in whom aPL dosages were available, the prevalence of persistent positivity (2 positive dosages spaced more than 12 weeks apart) was 21%.

Interestingly, a third of the cohort presented an antibody profile that did not allow them to be classified according to the classification criteria in force. This group corresponds to patients with a single positive dosage, either not recontrolled or recontrolled negative. This group was called Fluctuating. This fluctuating group was associated with arteriovenous fistula complications in a 2019 study.

Endothelial dysfunction is also implicated in the pathophysiology of APS. In clinical practice, the "flow mediated dilation" (FMD) test makes it possible to assess endothelial dysfunction in vivo. It involves the phenomenon of post-occlusive hyperemia which is mainly linked to NO and endothelium-dependent vasodilation. In the brachial artery, NO is the sole mediator of FMD. Endothelial dysfunction according to FMD has been described in populations with advanced chronic kidney disease, as well as patients with cardiovascular diseases. Hemodialysis patients with delayed/absence of arteriovenous fistula maturation have more pathological FMDs compared to dialysis patients without fistula problems. However, the additive role of aPL in this different population has not been studied in terms of endothelial dysfunction by FMD.

The objective of this study is to evaluate the weight of antiphospholipid biology on endothelial dysfunction in hemodialysis patients, using the FMD test.

1. Compare endothelial dysfunction by FMD according to the stage of chronic kidney disease and in comparison to a control group without chronic kidney disease.

2. Characterize the FMD pre or post dialysis and according to the duration of the long (for example between Thursday and Sunday) vs. short (between Tuesday and Thursday) inter-dialytic period.

3. Evaluate the relationship between endothelial dysfunction according to FMD, aPL positivity and arteriovenous fistula complications in hemodialysis patients.

4. Evaluate the risk factors associated with endothelial dysfunction according to FMD, and in particular evaluate the impact of antiphospholipid antibodies.

5. Evaluate the correlation between endothelial dysfunction according to FMD and other markers of endothelial dysfunction (urinary NO and metabolites of urinary NO, PGI2, endothelin, PGH2).

Detailed Description

Not available

Recruitment & Eligibility

Status
RECRUITING
Sex
All
Target Recruitment
200
Inclusion Criteria
  • Patients with Chronic kidney disease from stage G3a to G5
  • Healthy volunteers
Exclusion Criteria
  • Patients with chronic kidney disease stage G5 with no dosage available of antiphospholipid antibodies.

Study & Design

Study Type
INTERVENTIONAL
Study Design
PARALLEL
Arm && Interventions
GroupInterventionDescription
Chronic kidney disease at stage G4Flow mediated dilatation testRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) between 30 and 15 ml/min/1.73m²
Chronic kidney disease at stage G5 with dialyzeUrine samplingPatients who undergo dialyze
Chronic kidney disease at stage G4Urine samplingRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) between 30 and 15 ml/min/1.73m²
Chronic kidney disease at stage G4Blood samplingRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) between 30 and 15 ml/min/1.73m²
Chronic kidney disease at stage G3bFlow mediated dilatation testRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) between 45 and 30 ml/min/1.73m²
Chronic kidney disease at stage G3aUrine samplingRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) between 60 and 45 ml/min/1.73m²
Chronic kidney disease at stage G3aBlood samplingRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) between 60 and 45 ml/min/1.73m²
Chronic kidney disease at stage G3aFlow mediated dilatation testRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) between 60 and 45 ml/min/1.73m²
Chronic kidney disease at stage G5 with dialyzeBlood samplingPatients who undergo dialyze
ControlUrine samplingHealthy volunteer patient, without existing kidney disease
ControlFlow mediated dilatation testHealthy volunteer patient, without existing kidney disease
Chronic kidney disease at stage G3bUrine samplingRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) between 45 and 30 ml/min/1.73m²
Chronic kidney disease at stage G3bBlood samplingRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) between 45 and 30 ml/min/1.73m²
Chronic kidney disease at stage G5 not dialyzedUrine samplingRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) less than 15 ml/min/1.73m² but not dialyzed
Chronic kidney disease at stage G5 with dialyzeFlow mediated dilatation testPatients who undergo dialyze
Chronic kidney disease at stage G5 not dialyzedBlood samplingRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) less than 15 ml/min/1.73m² but not dialyzed
ControlBlood samplingHealthy volunteer patient, without existing kidney disease
Chronic kidney disease at stage G5 not dialyzedFlow mediated dilatation testRenal clearance according to CKD-EPI (Chronic Kidney Disease Epidemiology Collaboration) less than 15 ml/min/1.73m² but not dialyzed
Primary Outcome Measures
NameTimeMethod
Flow mediated dilatation test result (%)pre-intervention

Flow-mediated dilation (FMD) is a non-invasive vascular function test that measures the change in artery diameter in response to reactive hyperemia. The result of the test is expressed as a percentage.

E-Selectine plasma levelpre-intervention

E-Selectine plasma level

Nitric oxide (NO) urine concentrationpre-intervention

Nitric oxide (NO) urine concentration

Endothelin 1 (ET-1) urine concentrationpre-intervention

Endothelin 1 (ET-1) urine concentration

Endothelin 1 (ET-1) plasma levelpre-intervention

Endothelin 1 (ET-1) plasma level

P-Selectine plasma levelpre-intervention

P-Selectine plasma level

Intercellular Adhesion Molecule 1 (ICAM-1) plasma levelpre-intervention

Intercellular Adhesion Molecule 1 (ICAM-1) plasma level

Tumour Necrosis Factor alpha (TNF alpha) urine concentrationpre-intervention

Tumour Necrosis Factor alpha (TNF alpha) urine concentration

Nitric oxide (NO) plasma levelpre-intervention

Nitric oxide (NO) plasma level

Interleukin 6 (IL-6) plasma levelpre-intervention

Interleukin 6 (IL-6) plasma level

Interleukin 6 (IL-6) urine concentrationpre-intervention

Interleukin 6 (IL-6) urine concentration

Secondary Outcome Measures
NameTimeMethod

Trial Locations

Locations (1)

Brugmann University Hospital

🇧🇪

Brussels, Belgium

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