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Effect on metabolic acidosis due to hyperammonemia of high versus low intake of amino acid in VLBW infants.

Active, not recruiting
Conditions
At birth, a VLBW infant is abruptly disconnected from the ideal source of parenteralnutrition - the placenta. If the goal of post-natal nutrition in the VLBW infant is to mimic in utero nutrition. The VLBW infant should be immediately placed on balanced parenteral nutrition. In current praxis parenteral nutrition used to be delayed for several days.
MedDRA version: 9.1Level: LLTClassification code 10063258Term: Amino acid level
Registration Number
EUCTR2006-006440-55-DE
Lead Sponsor
niversity Childrin's Hospital
Brief Summary

Not available

Detailed Description

Not available

Recruitment & Eligibility

Status
Authorised-recruitment may be ongoing or finished
Sex
All
Target Recruitment
Not specified
Inclusion Criteria

Preterm infant with a birth weight below 2000g
Are the trial subjects under 18? yes
Number of subjects for this age range:
F.1.2 Adults (18-64 years) no
F.1.2.1 Number of subjects for this age range
F.1.3 Elderly (>=65 years) no
F.1.3.1 Number of subjects for this age range

Exclusion Criteria

Inborn error of metabolism which requires a reduction of amino acid intake

Study & Design

Study Type
Interventional clinical trial of medicinal product
Study Design
Not specified
Primary Outcome Measures
NameTimeMethod
Main Objective: Incidence of metabolic acidosis due to hyperamonemia with intake of 1.5 g/kg/d and increase to 3,5 g/kg/d amino acid from day 1 to day 5 of life in comparison to 3.5 g/kg/d amino acid at day 1 of life.;Secondary Objective: Comparison of extracellular water, total body water, energy expenditure, and protein and amino acid metabolism in the low and high amino acid group..<br>Examination of growth (weight gain, linear and head growth) until hospital discharge.;Primary end point(s): base excess due to hyperammonemia
Secondary Outcome Measures
NameTimeMethod
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